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Glycosylation of the HA protein of H5N1 virus increases its virulence in mice by exacerbating the host immune response
PubMed Full text in PMC Similar studies Analyze with GEO2R
Expression data of lungs of mice infected with two highly pathogenic avian influenza viruses
The 1918 PB2 protein, not HA, enhances the virulence of an avian influenza virus closely related to the 1918 pandemic virus through the inhibition of wnt signaling.
Disease severity is associated with differential gene expression at the early and late phases of infection in non-human primates infected with different H5N1 highly pathogenic avian influenza viruses
Viral determinants in H5N1 influenza A virus enable productive infection of HeLa cells
PubMed Full text in PMC Similar studies Analyze with GEO2RSRA Run Selector
Early and sustained innate immune response defines pathology and death in nonhuman primates infected by highly pathogenic influenza virus.
Expression data of lungs from chickens infected with different H5N1 Avian Influenza Viruses
Expression data of lungs from chickens infected with different H5N1 Avian Influenza Viruses [24 hours post infection]
Expression data of lungs from chickens infected with different H5N1 Avian Influenza Viruses [12 hours post infection]
Differences in cytokine production in human macrophages and in virulence in mice are attributable to the PA protein of H5N1 influenza viruses
RNA-seq analysis of miR-324-5p overexpression upon H5N1 infection in A549 cells
Characterizing the transcriptomic response of mice infected with A/Anhui/01/2013 (H7N9), A/Netherlands/219/2003 (H7N7), and A/Vietnam/1203/2004 (H5N1), and a pandemic H1N1 human virus, A/Mexico/4482/2007 (H1N1)
IM002, IM009 - Implication of inflammatory macrophages, nuclear receptors and interferon regulatory factors in increased virulence of pandemic 2009 H1N1 influenza A virus after host adaptation
Acute lung injury results from innate sensing of viruses by an ER stress pathway
Influenza A/Hong Kong/156/1997(H5N1) virus NS1 gene mutations F103L and M106I both increase IFN antagonism, virulence and cytoplasmic localization but differ in binding to RIG-I and CPSF30
Kidney epithelium cell line response to Influenza A virus (H3N2) non-structural 1 gene mutations
PubMed Full text in PMC Similar studies GEO Profiles Analyze DataSet
Lung epithelium cell line response to Influenza A virus (H3N2) non-structural 1 gene mutations
Host Regulatory Network Response to Infection with Highly Pathogenic H5N1 Avian Influenza Virus
Highly Pathogenic Avian Influenza Viruses Avoid Effective Inflammatory Response of Human Macrophages
An H5N1 virus-encoded atypical microRNA is a major contributor to cytokine storm and mortality
PubMed Full text in PMC Similar studies SRA Run Selector
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