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Expression profile of hematopoietic stem and progenitor cell (HSPC) compartment of FLT3-ITD and FLT3-ITD miR-155-/- mice
PubMed Full text in PMC Similar studies SRA Run Selector
Gene expression profile of Sca-1+ versus Sca-1- endothelial cells selected from the bone marrow of normal B6 mice
CircMYBL2 Regulates FLT3-ITD FLT3AML Translation in AML
PubMed Full text in PMC Similar studies Analyze with GEO2RSRA Run Selector
Dnmt3a haploinsufficiency transforms Flt3-ITD myeloproliferative disease into a rapid, spontaneous, and fully-penetrant acute myeloid leukemia
PubMed Full text in PMC Similar studies
Dnmt3a haploinsufficiency transforms Flt3-ITD myeloproliferative disease into a rapid, spontaneous, and fully-penetrant acute myeloid leukemia (Single Cell RNA-Seq)
Dnmt3a haploinsufficiency transforms Flt3-ITD myeloproliferative disease into a rapid, spontaneous, and fully-penetrant acute myeloid leukemia (Bulk RNA-Seq)
FLT3 inhibition upregulates HDAC8 via FOXO to inactivate p53 and promote maintenance of FLT3-ITD+ acute myeloid leukemia
PubMed Similar studies Analyze with GEO2RSRA Run Selector
FLT3-ITD mutation expands dendritic cells and alters CD4+ T cells in acute myeloid leukemia
Fetal and neonatal hematopoietic progenitors are functionally and transcriptionally resistant to Flt3-ITD mutations.
PubMed Full text in PMC Similar studies Analyze with GEO2R
Role of NFATc1 in patients with FLT3-ITD AML
PubMed Similar studies Analyze with GEO2R
Expression data from KSL and GMP cells of FLT3/ITD and FLT3/ITD-SmoM2 mice
FLT3/ITD-SmoM2 acute myeloid leukemia model: sorted bone marrow cells
PubMed Full text in PMC Similar studies GEO Profiles Analyze DataSet
GATM-Mediated Creatine Biosynthesis Enables Maintenance of FLT3-ITD-Mutant Acute Myeloid Leukemia
Autophagy inhibition impairs leukemia stem cell function in FLT3-ITD AML but has antagonistic interactions with tyrosine kinase inhibition
Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid leukaemia
PubMed Similar studies
Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid leukaemia [MV411_OCI-AML3_DMSO_AC220_24hr]
Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid leukaemia [iFLT3-ITD-Depletion_In VIVO]
PubMed Similar studies SRA Run Selector
Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid leukaemia [iFLT3-ITD-Depletion_In VITRO]
Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid leukaemia [MV411_OCI-AML3_ATF4_RNAP2_CHIP]
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