|
Status |
Public on Feb 26, 2019 |
Title |
PTENα/β paradoxically promote carcinogenesis through WDR5-H3K4me3 axis |
Organism |
Homo sapiens |
Experiment type |
Genome binding/occupancy profiling by high throughput sequencing Expression profiling by high throughput sequencing
|
Summary |
This SuperSeries is composed of the SubSeries listed below.
|
|
|
Overall design |
Refer to individual Series
|
|
|
Citation(s) |
31685992 |
|
Submission date |
Feb 24, 2019 |
Last update date |
Nov 14, 2019 |
Contact name |
ge mengkai |
E-mail(s) |
gemengk@sjtu.edu.cn
|
Phone |
15201867201
|
Organization name |
Shanghai Jiao Tong University School of Medicine (SJTU-SM)
|
Street address |
shanghai
|
City |
shanghai |
State/province |
State... |
ZIP/Postal code |
13185541251gmk |
Country |
China |
|
|
Platforms (1) |
GPL24676 |
Illumina NovaSeq 6000 (Homo sapiens) |
|
Samples (21)
|
|
This SuperSeries is composed of the following SubSeries:
|
GSE126982 |
PTENα/β paradoxically promote carcinogenesis through WDR5-H3K4me3 axis [ChIP-seq] |
GSE126983 |
PTENα/β paradoxically promote carcinogenesis through WDR5-H3K4me3 axis [RNA-seq] |
GSE135477 |
PTENa/b paradoxically promote carcinogenesis through WDR5-H3K4me3 axis [RNA-seq 2] |
GSE135478 |
PTENa/b paradoxically promote carcinogenesis through WDR5-H3K4me3 axis [ChIP-seq 2] |
|
Relations |
BioProject |
PRJNA524001 |