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Transcriptional elongation control of hypoxic response and therapeutic approaches
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RNA Polymerase II-associated factor 1 regulates the release and phosphorylation of paused RNA Polymerase II
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A positive feedback loop links opposing functions of P-TEFb/Cdk9 and histone H2B mono-ubiquitylation to regulate transcript elongation in fission yeast
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Distinct roles of Brd2 and Brd4 in potentiating the transcriptional program for Th17 cell differentiation
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Knockdown of Brd4 or SEC affects the HMBA-induced global Pol II pausing release
hnRNP R negatively regulates transcription by modulating the association of P-TEFb with 7SK and BRD4
HIF-1 Recruits TRIM28 and DNA-PK to Release Paused RNA Polymerase II and Activate Target Gene Transcription in Response to Hypoxia
Distinct Cdk9-phosphatase switches act on elongation factor Spt5 at the beginning and end of the RNA polymerase II transcription cycle
Ubiquitin-dependent turnover of MYC promotes loading of the PAF complex on RNA Polymerase II to drive transcriptional elongation
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Ubiquitin-dependent turnover of MYC promotes loading of the PAF complex on RNA Polymerase II to drive transcriptional elongation (ChIP-seq)
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Ubiquitin-dependent turnover of MYC promotes loading of the PAF complex on RNA Polymerase II to drive transcriptional elongation (RNA-seq)
PubMed Similar studies Analyze with GEO2RSRA Run Selector
JMJD5 couples with CDK9 to release the paused RNA polymerase II
Genome-wide map of RNA polymerase II, NELF, and DSIF in HeLa cells
Distinct layers of BRD4-PTEFb reveal bromodomain-independent function in transcriptional regulation
CDK8 ChIP-seq from HCT116 cells in normoxia and hypoxia
GRO-seq from HCT116 cells
Expression data from HCT116 colorectal cancer cells.
RNAPol2 accounts for tumor cells liability to JQ1
RNAPol2 accounts for tumor cells liability to JQ1 [ChIP-Seq]
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