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Sample GSM1010876 Query DataSets for GSM1010876
Status Public on Sep 28, 2012
Title HudsonAlpha_ChipSeq_HepG2_MYBL2_(SC-81192)_v042211.1
Sample type SRA
 
Source name HepG2
Organism Homo sapiens
Characteristics datatype: ChipSeq
datatype description: Chromatin IP Sequencing
antibody antibodydescription: Mouse monoclonal IgG, raised against a recombinant protein corresponding to an internal region of B-Myb of human origin. Antibody Target: MYBL2
antibody targetdescription: The protein encoded by this gene, a member of the MYB family of transcription factor genes, is a nuclear protein involved in cell cycle progression. The encoded protein is phosphorylated by cyclin A/cyclin-dependent kinase 2 during the S-phase of the cell cycle and possesses both activator and repressor activities. It has been shown to activate the cell division cycle 2, cyclin D1, and insulin-like growth factor-binding protein 5 genes. Transcript variants may exist for this gene, but their full-length natures have not been determined. (provided by RefSeq)
antibody vendorname: Santa Cruz Biotechnology
antibody vendorid: sc-81192
controlid: SL6019
labexpid: SL7564,SL7101
softwareversion: MACS
cell sex: M
antibody: MYBL2_(SC-81192)
antibody antibodydescription: Mouse monoclonal IgG, raised against a recombinant protein corresponding to an internal region of B-Myb of human origin. Antibody Target: MYBL2
antibody targetdescription: The protein encoded by this gene, a member of the MYB family of transcription factor genes, is a nuclear protein involved in cell cycle progression. The encoded protein is phosphorylated by cyclin A/cyclin-dependent kinase 2 during the S-phase of the cell cycle and possesses both activator and repressor activities. It has been shown to activate the cell division cycle 2, cyclin D1, and insulin-like growth factor-binding protein 5 genes. Transcript variants may exist for this gene, but their full-length natures have not been determined. (provided by RefSeq)
antibody vendorname: Santa Cruz Biotechnology
antibody vendorid: sc-81192
treatment: None
treatment description: No special treatment or protocol applies
protocol: v042211.1
protocol description: Faster ChIP protocol & AMpure XP size selection for ChIP-seq (Myers)
controlid: SL6019
labexpid: SL7564,SL7101
replicate: 1,2
softwareversion: MACS
Biomaterial provider ATCC
Treatment protocol None
Growth protocol HepG2_protocol.pdf
Extracted molecule genomic DNA
Extraction protocol Instrument model unknown. ("Illumina Genome Analyzer" specified by default). For more information, see http://genome.ucsc.edu/cgi-bin/hgTrackUi?db=hg19&g=wgEncodeHaibTfbs
 
Library strategy ChIP-Seq
Library source genomic
Library selection ChIP
Instrument model Illumina Genome Analyzer
 
Data processing http://genome.ucsc.edu/cgi-bin/hgTrackUi?db=hg19&g=wgEncodeHaibTfbs
 
Submission date Sep 28, 2012
Last update date May 15, 2019
Contact name ENCODE DCC
E-mail(s) encode-help@lists.stanford.edu
Organization name ENCODE DCC
Street address 300 Pasteur Dr
City Stanford
State/province CA
ZIP/Postal code 94305-5120
Country USA
 
Platform ID GPL9052
Series (2)
GSE32465 Transcription Factor Binding Sites by ChIP-seq from ENCODE/HAIB
GSE51334 DNA replication-timing boundaries separate stable chromosome domains with cell-type-specific functions
Relations
SRA SRX190332
BioSample SAMN01731761
Named Annotation GSM1010876_hg19_wgEncodeHaibTfbsHepg2Mybl2sc81192V0422111RawRep1.bigWig
Named Annotation GSM1010876_hg19_wgEncodeHaibTfbsHepg2Mybl2sc81192V0422111RawRep2.bigWig

Supplementary file Size Download File type/resource
GSM1010876_hg19_wgEncodeHaibTfbsHepg2Mybl2sc81192V0422111PkRep1.broadPeak.gz 404.5 Kb (ftp)(http) BROADPEAK
GSM1010876_hg19_wgEncodeHaibTfbsHepg2Mybl2sc81192V0422111PkRep2.broadPeak.gz 398.9 Kb (ftp)(http) BROADPEAK
GSM1010876_hg19_wgEncodeHaibTfbsHepg2Mybl2sc81192V0422111RawRep1.bigWig 28.6 Mb (ftp)(http) BIGWIG
GSM1010876_hg19_wgEncodeHaibTfbsHepg2Mybl2sc81192V0422111RawRep2.bigWig 23.6 Mb (ftp)(http) BIGWIG
SRA Run SelectorHelp
Raw data are available in SRA
Processed data provided as supplementary file

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