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Status |
Public on May 15, 2024 |
Title |
MATRIN3 deficiency leads to cGAS-STING activation (MinION RNA-Seq) |
Organism |
Homo sapiens |
Experiment type |
Expression profiling by high throughput sequencing
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Summary |
To investigate the role of Matrin 3 (MATR3) in innate immune response we analyzed and compared MATR3 KO Clones versus wildtype HAP1 cells.
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Overall design |
Comparative gene expression profiling analysis of MATR3 KO versus wildtype HAP1 cells using Oxford Nanopore long-read RNA-sequencing.
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Contributor(s) |
Islam Z, Chim B, Foroushani AK, Muljo S |
Citation(s) |
38712171 |
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Submission date |
Oct 28, 2022 |
Last update date |
Aug 01, 2024 |
Contact name |
Stefan Muljo |
E-mail(s) |
muljos@niaid.nih.gov
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Organization name |
NIH, NIAID
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Lab |
Laboratory of Immune System Biology
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Street address |
10 Center Drive
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City |
Bethesda |
State/province |
MD |
ZIP/Postal code |
20892-1892 |
Country |
USA |
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Platforms (1) |
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Samples (4)
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This SubSeries is part of SuperSeries: |
GSE216830 |
MATRIN3 deficiency leads to cGAS-STING activation |
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Relations |
BioProject |
PRJNA895453 |