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Links from GEO DataSets

Items: 15

1.
Full record GDS214

Duchenne muscular dystrophy (MuscleChip)

Examination of muscle biopsies from Duchenne muscular dystrophy patients and normal subjects of various age groups. Both mixed groups of patients (5 patient biopsies per group) and individual biopsies analyzed.
Organism:
Homo sapiens
Type:
Expression profiling by array, count, 4 age, 2 disease state sets
Platform:
GPL246
Series:
GSE465
30 Samples
Download data: CEL
2.

Expression profiling in the muscular dystrophies

(Submitter supplied) This is a large series human Duchenne muscular dystrophy patient muscle biopsies, in specific age groups, using all available Affymetrix arrays (including a custom MuscleChip produced by the Hoffman lab). Both mixed groups of patients (5 patient biopsies per group) and individual biopsies were done. Hypothesis: That the progression of DMD can be understood in terms of muscle molecular remodeling. Keywords: other
Organism:
Homo sapiens
Type:
Expression profiling by array
Datasets:
GDS214 GDS262 GDS264 GDS265 GDS270
7 related Platforms
57 Samples
Download data: CEL
Series
Accession:
GSE465
ID:
200000465
3.
Full record GDS270

Duchenne muscular dystrophy (HG-U95B)

Examination of muscle biopsies from Duchenne muscular dystrophy patients and normal subjects of various age groups. Both mixed groups of patients (5 patient biopsies per group) and individual biopsies analyzed.
Organism:
Homo sapiens
Type:
Expression profiling by array, count, 2 age, 2 disease state sets
Platform:
GPL92
Series:
GSE465
4 Samples
Download data: CEL
4.
Full record GDS265

Duchenne muscular dystrophy (HG-U95E)

Examination of muscle biopsies from Duchenne muscular dystrophy patients and normal subjects of various age groups. Both mixed groups of patients (5 patient biopsies per group) and individual biopsies analyzed.
Organism:
Homo sapiens
Type:
Expression profiling by array, count, 2 age, 2 disease state sets
Platform:
GPL95
Series:
GSE465
5 Samples
Download data: CEL
5.
Full record GDS264

Duchenne muscular dystrophy (HG-U95D)

Examination of muscle biopsies from Duchenne muscular dystrophy patients and normal subjects of various age groups. Both mixed groups of patients (5 patient biopsies per group) and individual biopsies analyzed.
Organism:
Homo sapiens
Type:
Expression profiling by array, count, 2 age, 2 disease state sets
Platform:
GPL94
Series:
GSE465
5 Samples
Download data: CEL
6.
Full record GDS262

Duchenne muscular dystrophy (HG-U95A)

Examination of muscle biopsies from Duchenne muscular dystrophy patients and normal subjects of various age groups. Both mixed groups of patients (5 patient biopsies per group) and individual biopsies analyzed.
Organism:
Homo sapiens
Type:
Expression profiling by array, count, 4 age, 2 disease state sets
Platform:
GPL91
Series:
GSE465
7 Samples
Download data: CEL
7.

Diaphram, comparison of wild type and mdx mice, 7 to 112 Days (Porter lab)

(Submitter supplied) Determination of gene expression changes in extraocular muscle of mdx (dystrophin-deficient) mice at postnatal ages 7, 14, 23, 28, 56, and 112 days. 3 independent replicates/age/strain. Data form part of publication: Human Molecular Genetics 13:257-269, 2004. Keywords = microarray Keywords = muscle Keywords: time-course
Organism:
Mus musculus
Type:
Expression profiling by array
Dataset:
GDS638
Platform:
GPL81
36 Samples
Download data: CEL
Series
Accession:
GSE1026
ID:
200001026
8.
Full record GDS638

Dystrophin-deficient mdx diaphram muscle development time course

Temporal analysis of diaphram muscle from dystrophin-deficient mdx mice, a Duchenne muscular dystrophy (DMD) model. Postnatal ages 7 to 112 days examined. Results provide insight into mechanisms of muscular dystrophy pathogenesis.
Organism:
Mus musculus
Type:
Expression profiling by array, count, 6 age, 2 strain sets
Platform:
GPL81
Series:
GSE1026
36 Samples
Download data: CEL
DataSet
Accession:
GDS638
ID:
638
9.

Mouse Models for Muscular Dystrophy

(Submitter supplied) Comparative analysis of gene expression levels from hindlimb muscle tissue from 8 week old mouse models for muscular dystrophy. We have used mouse models with dystrophin-, sarcoglycan-, sarcospan-, or dysferlin-deficiency. Keywords = muscular dystrophy Keywords: other
Organism:
Mus musculus
Type:
Expression profiling by array
Platform:
GPL1770
26 Samples
Download data
Series
Accession:
GSE2112
ID:
200002112
10.

Extraocular and hindlimb muscle, comparison of wild type and mdx mice, 56 days (Porter lab)

(Submitter supplied) Determination of gene expression changes in extraocular and hindlimb (gastrocnemius/soleus) of mdx (dystrophin-deficient) mice at postnatal day 56. 5 independent replicates/muscle group/strain. Keywords: parallel sample
Organism:
Mus musculus
Type:
Expression profiling by array
Dataset:
GDS703
Platform:
GPL32
20 Samples
Download data: CEL
Series
Accession:
GSE1472
ID:
200001472
11.
Full record GDS703

Dystrophin-deficient mdx extraocular and leg muscle

Analysis of extraocular (EOM) and hindlimb (gastrocnemius/soleus) muscle in mdx (dystrophin-deficient; Duchenne muscular dystrophy model) mice at postnatal day 56.5. Highly specific changes observed between dystrophic (leg) and spared (EOM) muscle.
Organism:
Mus musculus
Type:
Expression profiling by array, count, 2 strain, 2 tissue sets
Platform:
GPL32
Series:
GSE1472
20 Samples
Download data: CEL
DataSet
Accession:
GDS703
ID:
703
12.

fhl2b expression ameliorates muscular dystrophy

(Submitter supplied) This SuperSeries is composed of the SubSeries listed below.
Organism:
Danio rerio
Type:
Expression profiling by high throughput sequencing
Platform:
GPL24995
40 Samples
Download data: TAB
Series
Accession:
GSE242137
ID:
200242137
13.

fhl2b expression ameliorates muscular dystrophy [5 months EOM and trunk]

(Submitter supplied) In muscle dystrophies, muscle fibers loose integrity and die, leading to significant suffering and a shorter life. Strikingly, the extraocular muscles (EOMs), controlling eye movements, are spared and function well despite the disease progression. Although EOMs have been shown to have important differences compared to body musculature the mechanisms underlying this inherent resistance to muscle dystrophies remain largely unknown. more...
Organism:
Danio rerio
Type:
Expression profiling by high throughput sequencing
Platform:
GPL24995
12 Samples
Download data: TAB
Series
Accession:
GSE242136
ID:
200242136
14.

fhl2b expression ameliorates muscular dystrophy [5dpf trunk]

(Submitter supplied) In muscle dystrophies, muscle fibers loose integrity and die, leading to significant suffering and a shorter life. Strikingly, the extraocular muscles (EOMs), controlling eye movements, are spared and function well despite the disease progression. Although EOMs have been shown to have important differences compared to body musculature the mechanisms underlying this inherent resistance to muscle dystrophies remain largely unknown. more...
Organism:
Danio rerio
Type:
Expression profiling by high throughput sequencing
Platform:
GPL24995
16 Samples
Download data: TAB
Series
Accession:
GSE242135
ID:
200242135
15.

fhl2b expression ameliorates muscular dystrophy [20 months EOM and trunk]

(Submitter supplied) In muscle dystrophies, muscle fibers loose integrity and die, leading to significant suffering and a shorter life. Strikingly, the extraocular muscles (EOMs), controlling eye movements, are spared and function well despite the disease progression. Although EOMs have been shown to have important differences compared to body musculature the mechanisms underlying this inherent resistance to muscle dystrophies remain largely unknown. more...
Organism:
Danio rerio
Type:
Expression profiling by high throughput sequencing
Platform:
GPL24995
12 Samples
Download data: TAB
Series
Accession:
GSE242134
ID:
200242134
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