U.S. flag

An official website of the United States government

Display Settings:

Format
Sort by

Send to:

Choose Destination

Links from Nucleotide

Items: 4

1.
Smarcal1-Mediated Fork Reversal Triggers Mre11-Dependent Degradation of Nascent DNA in the Absence of Brca2 and Stable Rad51 Nucleofilaments
Arun Mouli Kolinjivadi, Vincenzo Sannino, Anna De Antoni, Karina Zadorozhny, Mairi Kilkenny, Hervé Técher, Giorgio Baldi, Rong Shen, Alberto Ciccia, Luca Pellegrini, Lumir Krejci, Vincenzo Costanzo
Mol Cell. 2017 Sep 7; 67(5): 867–881.e7. doi: 10.1016/j.molcel.2017.07.001
PMCID:
PMC5594205
AbstractArticle
2.
Rad51 protects nascent DNA from Mre11 dependent degradation and promotes continuous DNA synthesis
Yoshitami Hashimoto, Arnab Ray Chaudhuri, Massimo Lopes, Vincenzo Costanzo
Nat Struct Mol Biol. Author manuscript; available in PMC 2015 Jan 26.
Published in final edited form as: Nat Struct Mol Biol. 2010 Nov; 17(11): 1305–1311. Published online 2010 Oct 10. doi: 10.1038/nsmb.1927
PMCID:
PMC4306207
AbstractArticlePDF–1.3M
3.
RAD51 and MRE11 dependent reassembly of uncoupled CMG helicase complex at collapsed replication forks
Yoshitami Hashimoto, Fabio Puddu, Vincenzo Costanzo
Nat Struct Mol Biol. Author manuscript; available in PMC 2015 Jan 26.
Published in final edited form as: Nat Struct Mol Biol. 2012 Jan; 19(1): 17–24. Published online 2011 Dec 4. doi: 10.1038/nsmb.2177
PMCID:
PMC4306020
AbstractArticlePDF–955K
4.
Cdk1 uncouples CtIP-dependent resection and Rad51 filament formation during M-phase double-strand break repair
Shaun E. Peterson, Yinyin Li, Brian T. Chait, Max E. Gottesman, Richard Baer, Jean Gautier
J Cell Biol. 2011 Sep 5; 194(5): 705–720. doi: 10.1083/jcb.201103103
PMCID:
PMC3171114
AbstractArticlePDF–2.8M

Display Settings:

Format
Sort by

Send to:

Choose Destination

Supplemental Content

Filter your results:

Find related data

Recent activity

Clear Turn Off Turn On

Your browsing activity is empty.

Activity recording is turned off.

Turn recording back on

See more...
You are here: NCBI > Literature > PubMed Central (PMC)
Support Center