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Status |
Public on Dec 22, 2013 |
Title |
Epstein Barr Virus Nuclear Antigen 3C binds to BATF/IRF4/SPI1 sites and represses CDKN2A by recruiting Sin3A |
Organism |
Homo sapiens |
Experiment type |
Genome binding/occupancy profiling by high throughput sequencing
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Summary |
We report the application of ChIP Seq to study the Epstein Barr Virus Nuclear Antigen 3C, an essential transcriptional regulator involved in the transformation of Resting B Lymphocytes to the immortalized Lymphoblast Cell Lines
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Overall design |
Examination of viral and cellular transcription factors in 1 type of cell line
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Contributor(s) |
Jiang S, Willox B, Zhou H, Holthaus A, Wang A, Shi T, Maruo S, Johannsen E, Kharchenko P, Kieff E, Zhao B |
Citation(s) |
24344258 |
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Submission date |
Nov 21, 2013 |
Last update date |
May 15, 2019 |
Contact name |
Hufeng Zhou |
E-mail(s) |
williamzhoubwh@gmail.com
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Phone |
617-525-4263
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Organization name |
Harvard Medical School
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Department |
Department of Microbiology and Molecular Genetics
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Lab |
Kieff Lab
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Street address |
181 Longwood Ave,
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City |
Boston |
State/province |
M.A |
ZIP/Postal code |
02115 |
Country |
USA |
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Platforms (1) |
GPL11154 |
Illumina HiSeq 2000 (Homo sapiens) |
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Samples (2) |
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Relations |
BioProject |
PRJNA229507 |
SRA |
SRP033247 |
Supplementary data files not provided |
SRA Run Selector |
Raw data are available in SRA |
Processed data not provided for this record |
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