|
| Status |
Public on May 09, 2012 |
| Title |
DCK is frequently inactivated in acquired gemcitabine-resistant human cancer cells. |
| Organism |
Homo sapiens |
| Experiment type |
Expression profiling by array
|
| Summary |
We examined the microarray-based expression profiles of the cells to identify genes associated with gemcitabine resistance
|
| |
|
| Overall design |
Using three set of parental and resistant cell lines, PK-9 and RPK-9, PK-1 and RPK-1, and PK-59 and RPK-59, we studied mRNA transcription levels by microarray analysis to see if there are any of the key molecules for development of acquisition of the GEM resistance.
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| |
|
| Contributor(s) |
Saiki Y, Horii A |
| Citation(s) |
22490663 |
| |
| Submission date |
Jan 17, 2012 |
| Last update date |
Feb 22, 2018 |
| Contact name |
yuriko saiki |
| E-mail(s) |
ysaiki@molpath.med.tohoku.ac.jp
|
| Phone |
+81-22-717-8043
|
| Fax |
+81-22-717-8047
|
| Organization name |
Tohoku University School of Medicine
|
| Department |
Molecular Pathology
|
| Street address |
2-1 Seiryo-machi, Aoba-ku
|
| City |
Sendai |
| State/province |
Miyagi |
| ZIP/Postal code |
980-8575 |
| Country |
Japan |
| |
|
| Platforms (1) |
| GPL4133 |
Agilent-014850 Whole Human Genome Microarray 4x44K G4112F (Feature Number version) |
|
| Samples (12)
|
|
| Relations |
| BioProject |
PRJNA150705 |
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