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Status |
Public on May 06, 2024 |
Title |
FBXO32 drives pancreatic cancer progression |
Organism |
Homo sapiens |
Experiment type |
Expression profiling by high throughput sequencing
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Summary |
In order to profile the translation events that were regulated by FBXO32, Ribo-seq was performed on PANC-1 cells which was used to generate FBXO32 stably depleted pancreatic cancer cells.
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Overall design |
FBXO32-depleted or control PANC-1 cells were collected for ribosome profiling analysis to uncover potential downstream genes regulated by FBXO32 at the translational level. There were two bioreplicates for each group.
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Contributor(s) |
Su D, Zhang T |
Citation(s) |
38775804 |
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Submission date |
Mar 04, 2024 |
Last update date |
May 22, 2024 |
Contact name |
Dan Su |
E-mail(s) |
sudan6@mail2.sysu.edu.cn
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Phone |
86-13602452338
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Organization name |
Peking Union Medical college hospital
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Department |
Department of General Surgery
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Street address |
No. 1 Shuaifuyuan, Wangfujing Street, Beijing
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City |
Beijing |
ZIP/Postal code |
100730 |
Country |
China |
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Platforms (1) |
GPL24676 |
Illumina NovaSeq 6000 (Homo sapiens) |
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Samples (4)
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GSM8124743 |
PANC-1 cells, negative control, bioreplicate 1 |
GSM8124744 |
PANC-1 cells, negative control, bioreplicate 2 |
GSM8124745 |
PANC-1 cells, FBXO32 depeleted, bioreplicate 1 |
GSM8124746 |
PANC-1 cells, FBXO32 depeleted, bioreplicate 2 |
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Relations |
BioProject |
PRJNA1083527 |