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Status |
Public on Jul 08, 2019 |
Title |
A novel lncRNA lncRNA-AF339830 promotes colorectal carcinogenesis and glucose metabolism by stabilizing and specifying the transcription modification pattern of c-Myc [ChIP-Seq] |
Organism |
Homo sapiens |
Experiment type |
Genome binding/occupancy profiling by high throughput sequencing
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Summary |
To elucidate whether lncRNA-AF339830 plays a role in colorectal cancer tumorigenesis, a Chip-seq analysis was performed to compare the change of c-myc genome-wide binding of lncRNA-AF339830 siRNA and control siRNA transfectants.
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Overall design |
Chip-seq was performed in DLD1 colorectal cancer cells after lncRNA-AF339830 knock down
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Contributor(s) |
tingting Y |
Citation(s) |
31375671 |
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Submission date |
Jun 18, 2019 |
Last update date |
Aug 19, 2019 |
Contact name |
Haoyan Chen |
Organization name |
Renji Hospital
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Street address |
145 Shandong Road
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City |
Shanghai |
State/province |
Shanghai |
ZIP/Postal code |
200001 |
Country |
China |
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Platforms (1) |
GPL21290 |
Illumina HiSeq 3000 (Homo sapiens) |
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Samples (2) |
GSM3895624 |
Control siRNA (c-myc antibody treatment) |
GSM3895625 |
lncRNA-AF339830 siRNA (c-myc antibody treatment) |
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This SubSeries is part of SuperSeries: |
GSE132888 |
A novel lncRNA lncRNA-AF339830 promotes colorectal carcinogenesis and glucose metabolism by stabilizing and specifying the transcription modification pattern of c-Myc |
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Relations |
BioProject |
PRJNA549398 |
SRA |
SRP201716 |