| TRIM26 inhibits clear cell renal cell carcinoma progression through destabilizing ETK and thus inactivation of AKT/mTOR signaling. | TRIM26 inhibits clear cell renal cell carcinoma progression through destabilizing ETK and thus inactivation of AKT/mTOR signaling.
Zheng D, Ning J, Deng H, Ruan Y, Cheng F., Free PMC Article | 05/22/2024 |
| TRIM26 restricts Epstein-Barr virus infection in nasopharyngeal epithelial cells through K48-linked ubiquitination of HSP-90beta. | TRIM26 restricts Epstein-Barr virus infection in nasopharyngeal epithelial cells through K48-linked ubiquitination of HSP-90β.
Zhang M, Tan H, Gong Y, Faleti OD, Li D, Yang J, Huang J, Long J, Luo Q, Wu G, Zheng L, Lyu X. | 12/5/2023 |
| The E3 ligase TRIM26 suppresses ferroptosis through catalyzing K63-linked ubiquitination of GPX4 in glioma. | The E3 ligase TRIM26 suppresses ferroptosis through catalyzing K63-linked ubiquitination of GPX4 in glioma.
Wang Z, Xia Y, Wang Y, Zhu R, Li H, Liu Y, Shen N., Free PMC Article | 10/28/2023 |
| TRIM26 inhibited osteosarcoma progression through destabilizing RACK1 and thus inactivation of MEK/ERK signaling. | TRIM26 inhibited osteosarcoma progression through destabilizing RACK1 and thus inactivation of MEK/ERK signaling.
Xia K, Zheng D, Wei Z, Liu W, Guo W., Free PMC Article | 08/23/2023 |
| Tripartite Motif Containing 26 is a Positive Predictor for Endometrial Carcinoma Patients and Regulates Cell Survival in Endometrial Carcinoma. | Tripartite Motif Containing 26 is a Positive Predictor for Endometrial Carcinoma Patients and Regulates Cell Survival in Endometrial Carcinoma.
Lu T, Wu Y. | 12/24/2022 |
| Comprehensive analysis of expression profiles and prognosis of TRIM genes in human kidney clear cell carcinoma. | Comprehensive analysis of expression profiles and prognosis of TRIM genes in human kidney clear cell carcinoma.
Shen J, Wang R, Chen Y, Fang Z, Tang J, Yao J, Gao J, Zhou W, Chen X., Free PMC Article | 06/18/2022 |
| Inhibition of tripartite motif containing 26 inhibits non-small cell lung cancer cell growth. | Inhibition of tripartite motif containing 26 inhibits non-small cell lung cancer cell growth.
Xia YC, Zha JH. | 01/8/2022 |
| Competitive binding of E3 ligases TRIM26 and WWP2 controls SOX2 in glioblastoma. | Competitive binding of E3 ligases TRIM26 and WWP2 controls SOX2 in glioblastoma.
Mahlokozera T, Patel B, Chen H, Desouza P, Qu X, Mao DD, Hafez D, Yang W, Taiwo R, Paturu M, Salehi A, Gujar AD, Dunn GP, Mosammaparast N, Petti AA, Yano H, Kim AH., Free PMC Article | 12/11/2021 |
| Overexpression of tripartite motif containing 26 inhibits non-small cell lung cancer cell growth by suppressing PI3K/AKT signaling. | Overexpression of tripartite motif containing 26 inhibits non-small cell lung cancer cell growth by suppressing PI3K/AKT signaling.
Tao JL, Luo M, Sun H, Zhao HM, Sun QS, Huang ZM. | 07/17/2021 |
| TRIM26 Facilitates HSV-2 Infection by Downregulating Antiviral Responses through the IRF3 Pathway. | TRIM26 Facilitates HSV-2 Infection by Downregulating Antiviral Responses through the IRF3 Pathway.
Dhawan T, Zahoor MA, Heryani N, Workenhe ST, Nazli A, Kaushic C., Free PMC Article | 03/13/2021 |
| Knockdown of TRIM26 inhibits the proliferation, migration and invasion of bladder cancer cells through the Akt/GSK3beta/beta-catenin pathway. | Knockdown of TRIM26 inhibits the proliferation, migration and invasion of bladder cancer cells through the Akt/GSK3β/β-catenin pathway.
Xie X, Li H, Pan J, Han X. | 03/6/2021 |
| TRIM26 overexpression inhibited cell proliferation, migration, and invasion in Papillary thyroid carcinoma cells. TRIM26 overexpression also suppressed the epithelial-to-mesenchymal transition process. | Overexpression of TRIM26 suppresses the proliferation, metastasis, and glycolysis in papillary thyroid carcinoma cells.
Wang K, Chai L, Qiu Z, Zhang Y, Gao H, Zhang X. | 06/6/2020 |
| We have identified a novel gene TRIM26 and a novel SNP rs117565607_A associated with NPC risk by regulating transcriptional process and established a new functional link between TRIM26 downregulation and low immune response in NPC. | A regulatory mutant on TRIM26 conferring the risk of nasopharyngeal carcinoma by inducing low immune response.
Lyu XM, Zhu XW, Zhao M, Zuo XB, Huang ZX, Liu X, Jiang T, Yang XX, Li X, Long XB, Wang JG, Li JB, Han MY, Wang S, Liu TF, Zhang B, Sun T, Cheng Z, Qiu MC, Dong L, Zheng L, Zhang LC, Wang JH, Wei GG, Yao K, Wang Q, Zheng HF, Li X., Free PMC Article | 11/16/2019 |
| This paper demonstrates the importance of TRIM26 in regulating the cellular levels of NTH1, particularly under conditions of oxidative stress. | NTH1 Is a New Target for Ubiquitylation-Dependent Regulation by TRIM26 Required for the Cellular Response to Oxidative Stress.
Williams SC, Parsons JL., Free PMC Article | 06/22/2019 |
| Autoubiquitination of TRIM26 links TBK1 to NEMO in RLR-mediated innate antiviral immune response | Autoubiquitination of TRIM26 links TBK1 to NEMO in RLR-mediated innate antiviral immune response.
Ran Y, Zhang J, Liu LL, Pan ZY, Nie Y, Zhang HY, Wang YY. | 01/28/2017 |
| Down-regulation of TRIM26 is associated with worse prognosis of hepatocellular carcinoma. | TRIM26 functions as a novel tumor suppressor of hepatocellular carcinoma and its downregulation contributes to worse prognosis.
Wang Y, He D, Yang L, Wen B, Dai J, Zhang Q, Kang J, He W, Ding Q, He D. | 09/12/2015 |
| In addition to confirming one of the top findings in the meta-analysis, TRIM26, RNF5 and HLA-DRB3 have been identified as potential candidate genes for schizophrenia. | Expression QTL analysis of top loci from GWAS meta-analysis highlights additional schizophrenia candidate genes.
de Jong S, van Eijk KR, Zeegers DW, Strengman E, Janson E, Veldink JH, van den Berg LH, Cahn W, Kahn RS, Boks MP, Ophoff RA, PGC Schizophrenia (GWAS) Consortium., Free PMC Article | 12/29/2012 |
| The study reports no associations between polymorphisms in TRIM26 and the risk of aspirin-exacerbated respiratory disease in both logistic and regression analyses. | Association study between TRIM26 polymorphisms and risk of aspirin-exacerbated respiratory disease.
Lee JS, Bae JS, Kim JH, Kim JY, Park TJ, Pasaje CF, Park BL, Cheong HS, Jang AS, Uh ST, Park CS, Shin HD. | 07/21/2012 |
| Observational study of gene-disease association. (HuGE Navigator) | See all PubMed (2) articlesA major histocompatibility Class I locus contributes to multiple sclerosis susceptibility independently from HLA-DRB1*15:01.
Cree BA, Rioux JD, McCauley JL, Gourraud PA, Goyette P, McElroy J, De Jager P, Santaniello A, Vyse TJ, Gregersen PK, Mirel D, Hafler DA, Haines JL, Pericak-Vance MA, Compston A, Sawcer SJ, Oksenberg JR, Hauser SL, IMAGEN, IMSGC. High-density SNP screening of the major histocompatibility complex in systemic lupus erythematosus demonstrates strong evidence for independent susceptibility regions.
Barcellos LF, May SL, Ramsay PP, Quach HL, Lane JA, Nititham J, Noble JA, Taylor KE, Quach DL, Chung SA, Kelly JA, Moser KL, Behrens TW, Seldin MF, Thomson G, Harley JB, Gaffney PM, Criswell LA. | 01/20/2010 |