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SRX700326: GSM1503710: Mef2dIP_Mef2d_ChIP_WT_unstim_Ctx2; Mus musculus; ChIP-Seq
1 ILLUMINA (Illumina HiSeq 2000) run: 22.2M spots, 1.1G bases, 892.7Mb downloads

Submitted by: Gene Expression Omnibus (GEO)
Study: MEF2D drives photoreceptor development through a genome-wide competition for tissue-specific enhancers (ChIP-Seq)
show Abstracthide Abstract
Organismal development requires the precise coordination of genetic programs to regulate cell fate and function. MEF2 transcription factors play essential roles in this process but how these broadly expressed factors contribute to the generation of specific cell types during development is poorly understood. Here we show that despite being expressed in virtually all mammalian tissues, in the retina MEF2D binds to retina-specific enhancers and controls photoreceptor cell development. MEF2D achieves specificity by cooperating with a retina-specific factor CRX, which recruits MEF2D away from canonical MEF2 binding sites, and redirects it to retina-specific enhancers that lack the consensus MEF2 sequence. Once bound to retina-specific enhancers, MEF2D and CRX co-activate the expression of photoreceptor-specific genes that are critical for retinal function. These findings demonstrate that broadly expressed TFs acquire specific functions through competitive recruitment to enhancers by tissue-specific TFs, and through cooperative activation of these enhancers to regulate tissue-specific genes. Overall design: ChIP-seq for MEF2D in WT, Mef2d KO and Crx KO retinae and WT cultured cortical neurons. ChIP for H3K27Ac in WT and Mef2d KO retinae
Sample: Mef2dIP_Mef2d_ChIP_WT_unstim_Ctx2
SAMN03067474 • SRS700511 • All experiments • All runs
Organism: Mus musculus
Library:
Instrument: Illumina HiSeq 2000
Strategy: ChIP-Seq
Source: GENOMIC
Selection: ChIP
Layout: SINGLE
Construction protocol: MEF2D ChIP from mouse retinas was performed as previously described for brain tissue (Hong et al., 2008) with the following modifications: p11 mouse retinas were dissected in ice-cold HBSS prior to homogenization and crosslinking. 4μg of anti-MEF2D antibody was pre-bound to 15μl of Protein A dynabeads (Life Technologies) per immunoprecipitation (IP) from approximately 100 million retinal cells. H3K27Ac ChIP was performed as described above with the following modifications: 10 mM sodium butyrate was added to all solutions until post-IP washes with the exception of cross-linking buffer. Chromatin was fragmented for H3K27Ac ChIP by brief sonication followed by MNase (New England Biolabs) digestion for 8 minutes at 37C to generate mononucleosomes. 0.25μg of anti-H3K27Ac antibody was used per IP from 10 million retinal cells. After reverse crosslinking all samples were purified using phenol/chloroform/isoamyl alcohol followed by column clean up. Single-end short read sequencing libraires were prepared using standard protocols, following the manufacturers instructions. were prepared according to Illumina's instructions accompanying the DNA Sample Kit (Part# 0801-0303). Briefly, DNA was end-repaired using a combination of T4 DNA polymerase, E. coli DNA Pol I large fragment (Klenow polymerase) and T4 polynucleotide kinase. The blunt, phosphorylated ends were treated with Klenow fragment (32 to 52 exo minus) and dATP to yield a protruding 3- 'A' base for ligation of Illumina's adapters which have a single 'T' base overhang at the 3’ end. After adapter ligation DNA was PCR amplified with Illumina primers for 15 cycles and library fragments of ~250 bp (insert plus adaptor and PCR primer sequences) were band isolated from an agarose gel. The purified DNA was captured on an Illumina flow cell for cluster generation. Libraries were sequenced on the Genome Analyzer following the manufacturer's protocols.
Experiment attributes:
GEO Accession: GSM1503710
Links:
External link:
Runs: 1 run, 22.2M spots, 1.1G bases, 892.7Mb
Run# of Spots# of BasesSizePublished
SRR157450122,178,2091.1G892.7Mb2015-07-22

ID:
986123

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