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SRX4962155: GSM3453157: AGL7_low-dose antibiotics; Sus scrofa; Bisulfite-Seq
1 ILLUMINA (HiSeq X Ten) run: 73M spots, 20.9G bases, 10.1Gb downloads

Submitted by: NCBI (GEO)
Study: Multi-omics profiling highlights lipid metabolism alterations of pigs fed with low-dose antibiotics
show Abstracthide Abstract
Based on the gut-liver axis theory, to survey the effect of low-dose antibiotic (LDA) to hepatocellular functions of weight gain and metabolic imbalance by epigenetic transcription regulation, 32 weaned piglets were employed as the animal models and half were treated with Chlorotetracycline and Virginiamycin for four weeks. During the experiment, LDA showed an obviously growth-promoting effect, which was exemplified by the significantly elevated body weight and average daily gain. The DNA methylome profiling of promoter using liquid hybridization capture-based bisulfite sequencing (LHC-BS) indicated most of 745 differential methylation regions (DMRs) were hypermethylated in the LDA group, and some other DMRs were significantly enriched in genes related with fatty acids metabolic pathways, such as FABP1 and PCK1. Meanwhile, three key DEGs with the function of lipid metabolism and immunity were obtained by the liver strand-specific transcriptome analysis, including ALOX15, CXCL10 and NNMT, which were highly elevated for expressions in the LDA group. Correspondingly, the lipidome analyses of serum by LC-MS identified 38 significantly differential lipids, most of which were down-regulated in the LDA group. All these results suggest antibiotics could induce epigenetic and transcriptional changes of key genes related lipid metabolism pathway, and contributed to a more efficient metabolism of lipids. Overall design: 32 samples for ssRNA-seq,8 samples for LHC-BS
Sample: AGL7_low-dose antibiotics
SAMN10354883 • SRS4001486 • All experiments • All runs
Organism: Sus scrofa
Library:
Instrument: HiSeq X Ten
Strategy: Bisulfite-Seq
Source: GENOMIC
Selection: RANDOM
Layout: PAIRED
Construction protocol: For RNA-seq:RNAs were extracted by Trizol and quantified by Qubit®3.0 fluorometer (Thermo fisher scientific, Cat#Q33216). A total amount of 1.5 μg RNA per liver sample was used as input material for preparations. For Bisulfite-seq:DNAs of liver tissues from these piglet samples were extracted by DNeasy Blood & Tissue Kits (QIAGEN, Cat#69504) according to the manufactural instruments and quantified by Qubit®3.0 fluorometer (Thermo fisher scientific, Cat#Q33216). For RNA-seq:Sequencing libraries were constructed using NEBNext® UltraTM RNA library Prep Kit for Illumina® (NEB, USA) following manufacturer's recommendations. In brief, mRNA was purified from the total RNA using poly-T oligo-attached magnetic beads. Fragmentation was carried out using divalent cations under elevated temperature in NEBNext First Strand Synthesis Reaction Buffer (5×). First strand cDNA was synthesized using random hexamer primer and M-MuLV Reverse Transcriptase (RNaseH). Second strand cDNA synthesis was subsequently performed using DNA polymerase I and RNaseH, with the dUTP instead of deoxythymidine triphosphate (dTTP). Remaining overhangs were converted into blunt ends via exonuclease/polymerase activities. After adenylation of 3' ends of DNA fragments, NEBNext Adaptor with hairpin loop structure were ligated to prepare for hybridization. To select cDNA fragments with right length, the library fragments were purified with AMPure XP system (Beckman Coulter, Beverly, USA). Then 3 μl USER Enzyme (NEB, USA) was used with size-selected, adaptor-ligated cDNA at 37°C for 15 min followed by 5 min at 95°C before PCR, which allowed selective degradation of the second cDNA strand contained dUTP for the reservation of the transcript direction and strand-specific information. Then PCR was performed with Phusion High-Fidelity DNA polymerase, Universal PCR primers and Index (X) Primer. At last, products were purified (AMPure XP system) and library quality was assessed on the Agilent Bioanalyzer 2100 system. The clustering of the index-coded samples was performed on a cBot Cluster Generation System using HiSeq4000 PE Cluster Kit (Illumina) according to the manufacturer's instructions. For Bisulfite-seq: Briefly, 1 μg DNA of per sample was processed by fragmentation using Covaris, following with blunt end repair, 3′-adenylation, and 5′-methylcytosine index adapter ligation. Then, 250 ng DNA from each of three/four adapter-ligated libraries were pooled together for the liquid hybridization capture procedure. The hybridization probe was synthesized and purchased from Roche Nimblegen Incorporation. Finally, the captured DNA was eluted in 50 μl of 10 M NaOH with incubation at room temperature for 10 min. The supernatant was transferred into a new tube and neutralized with 50 μl of 10 M HAc and then purified using MiniElute PCR Purification Kit (QIAGEN, Cat#28004). For bisulfite conversion, 200 ng of unmethylated λDNA was added into each captured product and then ZYMO EZ DNA Methylation-Gold Kit™ (ZYMO, Cat#D5005) was employed to convert unmethylated cytosine into uracil according to the instructions. After purification, PCR was carried out with JumpStart™ Taq DNA Polymerase (Sigma, Cat#D9307) using the program of 94°C 30s, 15 cycles of 94°C 10 s, 60°C 30 s, 72°C 40s then prolong at 72°C for 5 min and hold at 12°C. The PCR products were purified using AMPure XP beads (Agencourt, Cat#A63881) and were quantified by the Agilent Bioanalyzer 2100 system (Agilent Technologies, CA, USA).
Experiment attributes:
GEO Accession: GSM3453157
Links:
Runs: 1 run, 73M spots, 20.9G bases, 10.1Gb
Run# of Spots# of BasesSizePublished
SRR814141372,991,66320.9G10.1Gb2019-10-31

ID:
6691170

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