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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1987 1
1988 1
1989 1
1990 1
1991 1
1992 5
1993 6
1994 6
1995 17
1996 10
1997 8
1998 10
1999 15
2000 11
2001 5
2002 12
2003 19
2004 13
2005 17
2006 14
2007 19
2008 26
2009 16
2010 13
2011 26
2012 29
2013 22
2014 28
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2017 34
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2020 16
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2023 20
2024 7

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493 results

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Page 1
Mechanisms of Renal Fibrosis.
Humphreys BD. Humphreys BD. Annu Rev Physiol. 2018 Feb 10;80:309-326. doi: 10.1146/annurev-physiol-022516-034227. Epub 2017 Oct 25. Annu Rev Physiol. 2018. PMID: 29068765 Review.
Tubulointerstitial fibrosis is a chronic and progressive process affecting kidneys during aging and in chronic kidney disease (CKD), regardless of cause. CKD and renal fibrosis affect half of adults above age 70 and 10% of the world's population. ...
Tubulointerstitial fibrosis is a chronic and progressive process affecting kidneys during aging and in chronic kidney disease …
Cellular and molecular mechanisms of renal fibrosis.
Liu Y. Liu Y. Nat Rev Nephrol. 2011 Oct 18;7(12):684-96. doi: 10.1038/nrneph.2011.149. Nat Rev Nephrol. 2011. PMID: 22009250 Free PMC article. Review.
Renal fibrosis, particularly tubulointerstitial fibrosis, is the common final outcome of almost all progressive chronic kidney diseases. Renal fibrosis is also a reliable predictor of prognosis and a major determinant of renal insufficiency. ...
Renal fibrosis, particularly tubulointerstitial fibrosis, is the common final outcome of almost all progressive chronic …
Tubulointerstitial nephritis: diagnosis, treatment, and monitoring.
Joyce E, Glasner P, Ranganathan S, Swiatecka-Urban A. Joyce E, et al. Pediatr Nephrol. 2017 Apr;32(4):577-587. doi: 10.1007/s00467-016-3394-5. Epub 2016 May 7. Pediatr Nephrol. 2017. PMID: 27155873 Free PMC article. Review.
Tubulointerstitial nephritis (TIN) is a frequent cause of acute kidney injury (AKI) that can lead to chronic kidney disease (CKD). TIN is associated with an immune-mediated infiltration of the kidney interstitium by inflammatory cells, which may progress to fibrosis …
Tubulointerstitial nephritis (TIN) is a frequent cause of acute kidney injury (AKI) that can lead to chronic kidney disease (CKD). TI …
IgG4-related disease: a clinical perspective.
Maritati F, Peyronel F, Vaglio A. Maritati F, et al. Rheumatology (Oxford). 2020 May 1;59(Suppl 3):iii123-iii131. doi: 10.1093/rheumatology/kez667. Rheumatology (Oxford). 2020. PMID: 32348524 Review.
Common presentations include major salivary and lacrimal gland enlargement, orbital disease, autoimmune pancreatitis, retroperitoneal fibrosis and tubulointerstitial nephritis. The main histopathological features are a dense, polyclonal, lymphoplasmacytic infiltrate …
Common presentations include major salivary and lacrimal gland enlargement, orbital disease, autoimmune pancreatitis, retroperitoneal fib …
Mitochondrial dysfunction in diabetic tubulopathy.
Yao L, Liang X, Qiao Y, Chen B, Wang P, Liu Z. Yao L, et al. Metabolism. 2022 Jun;131:155195. doi: 10.1016/j.metabol.2022.155195. Epub 2022 Mar 28. Metabolism. 2022. PMID: 35358497 Free article. Review.
Recently, the major focus of glomerular lesions of DKD has partly shifted to diabetic tubulopathy because of renal insufficiency and prognosis of patients is closely related to tubular atrophy and interstitial fibrosis. Indeed, the proximal tubule enriching in mitochondria …
Recently, the major focus of glomerular lesions of DKD has partly shifted to diabetic tubulopathy because of renal insufficiency and prognos …
Adenine-induced chronic kidney disease in rats.
Diwan V, Brown L, Gobe GC. Diwan V, et al. Nephrology (Carlton). 2018 Jan;23(1):5-11. doi: 10.1111/nep.13180. Nephrology (Carlton). 2018. PMID: 29030945 Review.
The original adenine diet model produced rapid-onset kidney disease with extensive tubulointerstitial fibrosis, tubular atrophy, crystal formation and marked vessel calcification. ...
The original adenine diet model produced rapid-onset kidney disease with extensive tubulointerstitial fibrosis, tubular atroph …
Autosomal dominant tubulointerstitial kidney disease.
Devuyst O, Olinger E, Weber S, Eckardt KU, Kmoch S, Rampoldi L, Bleyer AJ. Devuyst O, et al. Nat Rev Dis Primers. 2019 Sep 5;5(1):60. doi: 10.1038/s41572-019-0109-9. Nat Rev Dis Primers. 2019. PMID: 31488840 Review.
Autosomal dominant tubulointerstitial kidney disease (ADTKD) is a recently defined entity that includes rare kidney diseases characterized by tubular damage and interstitial fibrosis in the absence of glomerular lesions, with inescapable progression to end-stage ren …
Autosomal dominant tubulointerstitial kidney disease (ADTKD) is a recently defined entity that includes rare kidney diseases characte …
Innate immunity in diabetic kidney disease.
Tang SCW, Yiu WH. Tang SCW, et al. Nat Rev Nephrol. 2020 Apr;16(4):206-222. doi: 10.1038/s41581-019-0234-4. Epub 2020 Jan 15. Nat Rev Nephrol. 2020. PMID: 31942046 Review.
Toll-like receptors detect endogenous danger-associated molecular patterns generated during diabetes and induce a sterile tubulointerstitial inflammatory response via the NF-kappaB signalling pathway. ...The kallikrein-kinin system promotes inflammatory processes via the g …
Toll-like receptors detect endogenous danger-associated molecular patterns generated during diabetes and induce a sterile tubulointerstit …
Pathophysiological Mechanisms of Renal Fibrosis: A Review of Animal Models and Therapeutic Strategies.
Nogueira A, Pires MJ, Oliveira PA. Nogueira A, et al. In Vivo. 2017 Jan 2;31(1):1-22. doi: 10.21873/invivo.11019. In Vivo. 2017. PMID: 28064215 Free PMC article. Review.
Thus, one of the consequences of CKD is glomerulosclerosis and tubulointerstitial fibrosis caused by an imbalance between excessive synthesis and reduced breakdown of the ECM. ...However, there are currently no effective treatments for preventing the progression of …
Thus, one of the consequences of CKD is glomerulosclerosis and tubulointerstitial fibrosis caused by an imbalance between exce …
Diabetes and Kidney Disease: Role of Oxidative Stress.
Jha JC, Banal C, Chow BS, Cooper ME, Jandeleit-Dahm K. Jha JC, et al. Antioxid Redox Signal. 2016 Oct 20;25(12):657-684. doi: 10.1089/ars.2016.6664. Epub 2016 Apr 1. Antioxid Redox Signal. 2016. PMID: 26906673 Free PMC article. Review.
Critical Issues and Future Directions: In the present review, we summarize and critically analyze molecular and cellular mechanisms that have been demonstrated to be involved in NOX-induced renal injury in diabetes, with particular focus on the role of increased glomerular injury …
Critical Issues and Future Directions: In the present review, we summarize and critically analyze molecular and cellular mechanisms that hav …
493 results