Is C-reactive protein an innocent bystander or proatherogenic culprit? C-reactive protein promotes atherothrombosis

Circulation. 2006 May 2;113(17):2135-50; discussion 2150.
No abstract available

Publication types

  • Review
  • Comment

MeSH terms

  • Animals
  • Atherosclerosis / etiology*
  • Azides / toxicity
  • C-Reactive Protein / toxicity*
  • Endothelial Cells / drug effects
  • Endothelial Cells / physiology
  • Humans
  • Lipopolysaccharides / toxicity
  • Macrophages / drug effects
  • Matrix Metalloproteinases / metabolism
  • Mice
  • Monocytes / drug effects
  • Muscle, Smooth, Vascular / drug effects
  • Myocardial Infarction / etiology
  • NF-kappa B / physiology
  • Nitric Oxide Synthase Type III / antagonists & inhibitors
  • Receptor for Advanced Glycation End Products
  • Receptors, IgG / physiology
  • Receptors, Immunologic / physiology
  • Scavenger Receptors, Class E / physiology
  • Tumor Necrosis Factor-alpha / physiology
  • Tunica Intima / pathology

Substances

  • Azides
  • Lipopolysaccharides
  • NF-kappa B
  • OLR1 protein, human
  • Receptor for Advanced Glycation End Products
  • Receptors, IgG
  • Receptors, Immunologic
  • Scavenger Receptors, Class E
  • Tumor Necrosis Factor-alpha
  • C-Reactive Protein
  • Nitric Oxide Synthase Type III
  • Matrix Metalloproteinases