sodium channel protein type 5 subunit alpha isoform X1 [Homo sapiens]

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Knockdown of SCN5A alters metabolic-associated genes and aggravates hypertrophy in the cardiomyoblast. [Mol Biol Rep. 2024]
Knockdown of SCN5A alters metabolic-associated genes and aggravates hypertrophy in the cardiomyoblast.
Tariq U, Sarkar S, Malladi N, Kumar R, Bugga P, Chakraborty P, Banerjee SK. Mol Biol Rep. 2024 May 17; 51(1):661. Epub 2024 May 17.
Overexpression of SCN5A overcomes ABC transporter-mediated multidrug resistance in acute myeloid leukemia through promoting apoptosis. [Expert Rev Hematol. 2024]
Overexpression of SCN5A overcomes ABC transporter-mediated multidrug resistance in acute myeloid leukemia through promoting apoptosis.
Xu K, Zhuang XX, Shi XW. Expert Rev Hematol. 2024 Jan-Mar; 17(1-3):87-94. Epub 2024 Feb 2.
SCN5A-L256del and L1621F exhibit loss-of-function properties related to autosomal recessive congenital cardiac disorders presenting as sick sinus syndrome, dilated cardiomyopathy, and sudden cardiac death. [Gene. 2024]
SCN5A-L256del and L1621F exhibit loss-of-function properties related to autosomal recessive congenital cardiac disorders presenting as sick sinus syndrome, dilated cardiomyopathy, and sudden cardiac death.
Shi J, Pan X, Wang Z, Yi M, Xie S, Zhang X, Tao D, Yang Y, Liu Y. Gene. 2024 Mar 10; 898:148093. Epub 2023 Dec 19.

RefSeq genomic sequence
See the genomic reference sequence for the SCN5A gene (NG_008934.1).
RefSeq mRNA
See reference mRNA sequence for the SCN5A gene (XM_011533991.3).

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sodium channel protein type 5 subunit alpha isoform X1 [Homo sapiens]
sodium channel protein type 5 subunit alpha isoform X1 [Homo sapiens]
gi|767923970|ref|XP_011532293.1|

Protein
Dinitrochlorobenzene
Dinitrochlorobenzene
A skin irritant that may cause dermatitis of both primary and allergic types. Contact sensitization with DNCB has been used as a measure of cellular immunity. DNCB is also use...<br/>Year introduced: 1987(1975)

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Protein