Homo sapiens KRAS proto-oncogene, GTPase (KRAS), transcript variant a, - Nucleotide

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Epidemiological and clinicopathological features of KRAS, NRAS, BRAF mutations and MSI in Chinese patients with stage I-III colorectal cancer. [Medicine (Baltimore). 2024]
Epidemiological and clinicopathological features of KRAS, NRAS, BRAF mutations and MSI in Chinese patients with stage I-III colorectal cancer.
Wang W, Wang R, Han X, Zhang W, Zhu L, Gu Y. Medicine (Baltimore). 2024 Apr 5; 103(14):e37693.
STAT3 is a genetic modifier of TGF-beta induced EMT in KRAS mutant pancreatic cancer. [Elife. 2024]
STAT3 is a genetic modifier of TGF-beta induced EMT in KRAS mutant pancreatic cancer.
D'Amico S, Kirillov V, Petrenko O, Reich NC. Elife. 2024 Apr 4; 13. Epub 2024 Apr 4.
Circular RNAs in the KRAS pathway: Emerging players in cancer progression. [Pathol Res Pract. 2024]
Circular RNAs in the KRAS pathway: Emerging players in cancer progression.
Hussain MS, Moglad E, Afzal M, Bansal P, Kaur H, Deorari M, Ali H, Shahwan M, Hassan Almalki W, Kazmi I, et al. Pathol Res Pract. 2024 Apr; 256:155259. Epub 2024 Mar 11.

RefSeq alternative splicing
See 6 reference mRNA sequence splice variants for the KRAS gene.
RefSeq protein product
See the reference protein sequence for GTPase KRas isoform a (NP_203524.1).

Protein
Protein sequence from Nucleotide coding region
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Ensembl [Ensembl]
Ensembl
Ensembl [Ensembl]
Ensembl
KRAS [Guide to Pharmacology]
KRAS
Guide to Pharmacology
GenScript latest version of gene cDNA ORF Clone [GenScript latest version of g...]
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LRG_344t2 (0)
Nucleotide
SRA Links for BioSample (Select 13172) (12)
SRA
Homo sapiens alpha-2-macroglobulin (A2M), transcript variant 1, mRNA
Homo sapiens alpha-2-macroglobulin (A2M), transcript variant 1, mRNA
gi|1779421316|ref|NM_000014.6|

Nucleotide
Sunscreening Agents
Sunscreening Agents
Chemical or physical agents that protect the skin from sunburn and erythema by absorbing or blocking ultraviolet radiation.<br/>Year introduced: 1972(1966)

MeSH
Antimutagenic Agents
Antimutagenic Agents
Agents that reduce the frequency or rate of spontaneous or induced mutations independently of the mechanism involved.<br/>Year introduced: 1992

MeSH

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Nucleotide