Homo sapiens unc-93 homolog B1, TLR signaling regulator (UNC93B1), mRN - Nucleotide

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Gain-of-function human UNC93B1 variants cause systemic lupus erythematosus and chilblain lupus. [J Exp Med. 2024]
Gain-of-function human UNC93B1 variants cause systemic lupus erythematosus and chilblain lupus.
David C, Arango-Franco CA, Badonyi M, Fouchet J, Rice GI, Didry-Barca B, Maisonneuve L, Seabra L, Kechiche R, Masson C, et al. J Exp Med. 2024 Aug 5; 221(8). Epub 2024 Jun 13.
Genetic variants in UNC93B1 predispose to childhood-onset systemic lupus erythematosus. [Nat Immunol. 2024]
Genetic variants in UNC93B1 predispose to childhood-onset systemic lupus erythematosus.
Al-Azab M, Idiiatullina E, Liu Z, Lin M, Hrovat-Schaale K, Xian H, Zhu J, Yang M, Lu B, Zhao Z, et al. Nat Immunol. 2024 Jun; 25(6):969-980. Epub 2024 Jun 3.
Mitochondrial Raf1 Regulates Glutamine Catabolism. [bioRxiv. 2024]
Mitochondrial Raf1 Regulates Glutamine Catabolism.
Shanderson RL, Ferguson ID, Siprashvili Z, Ducoli L, Li AM, Miao W, Srinivasan S, Velasco MG, Li Y, Ye J, et al. bioRxiv. 2024 Mar 9; . Epub 2024 Mar 9.

RefSeq alternative splicing
See 5 reference mRNA sequence splice variants for the UNC93B1 gene.

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Protein sequence from Nucleotide coding region
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Nucleotide