Homo sapiens intraflagellar transport 140 (IFT140), mRNA

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Cullin 3 Exon 9 Deletion in Familial Hyperkalemic Hypertension Impairs Cullin3-Ring-E3 Ligase (CRL3) Dynamic Regulation and Cycling. [Int J Mol Sci. 2022]
Cullin 3 Exon 9 Deletion in Familial Hyperkalemic Hypertension Impairs Cullin3-Ring-E3 Ligase (CRL3) Dynamic Regulation and Cycling.
Kouranti I, Abdel Khalek W, Mazurkiewicz S, Loisel-Ferreira I, Gautreau AM, Pintard L, Jeunemaitre X, Clauser E. Int J Mol Sci. 2022 May 5; 23(9). Epub 2022 May 5.
Monoallelic IFT140 pathogenic variants are an important cause of the autosomal dominant polycystic kidney-spectrum phenotype. [Am J Hum Genet. 2022]
Monoallelic IFT140 pathogenic variants are an important cause of the autosomal dominant polycystic kidney-spectrum phenotype.
Senum SR, Li YSM, Benson KA, Joli G, Olinger E, Lavu S, Madsen CD, Gregory AV, Neatu R, Kline TL, et al. Am J Hum Genet. 2022 Jan 6; 109(1):136-156. Epub 2021 Dec 9.
Dual proteome-scale networks reveal cell-specific remodeling of the human interactome. [Cell. 2021]
Dual proteome-scale networks reveal cell-specific remodeling of the human interactome.
Huttlin EL, Bruckner RJ, Navarrete-Perea J, Cannon JR, Baltier K, Gebreab F, Gygi MP, Thornock A, Zarraga G, Tam S, et al. Cell. 2021 May 27; 184(11):3022-3040.e28. Epub 2021 May 6.

RefSeq alternative splicing
See 51 reference mRNA sequence splice variants for the IFT140 gene.
RefSeq protein product
See the reference protein sequence for intraflagellar transport protein 140 homolog (NP_055529.2).

Protein
Protein sequence from Nucleotide coding region
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OMIM
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PubMed (RefSeq)
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Nucleotide