|
Status |
Public on Jan 01, 2024 |
Title |
Lung, de novo SCLC, chromatin, rep 1 |
Sample type |
SRA |
|
|
Source name |
Lung
|
Organism |
Mus musculus |
Characteristics |
tissue: Lung genotype: PNEC-RPMT
|
Extracted molecule |
genomic DNA |
Extraction protocol |
Established Omni-ATAC extraction protocol Established Omni-ATAC library construction protocol
|
|
|
Library strategy |
ATAC-seq |
Library source |
genomic |
Library selection |
other |
Instrument model |
Illumina HiSeq 2500 |
|
|
Data processing |
Bulk ATAC-seq raw reads were processed using the standard nf-core ATAC-seq pipeline. Library quality was assessed using the nf-core quality-control report unique mapped reads, GC-content, and peak annotation. Loess normalization was used for differential accessibility testing to minimize technical batch effects. Assembly: mm10 Supplementary files format and content: .txt: comma separated text file. This contains the consensus peak annotations for ATAC chromatin peaks across all replicates.
|
|
|
Submission date |
Nov 19, 2023 |
Last update date |
Jan 01, 2024 |
Contact name |
Ashley M Laughney |
E-mail(s) |
ashley.laughney@gmail.com
|
Organization name |
Weill Cornell Medicine
|
Department |
Physiology, Biophysics, and Systems Biology
|
Lab |
Laughney Lab
|
Street address |
413 East 69th Street
|
City |
NEW YORK |
State/province |
New York |
ZIP/Postal code |
10021 |
Country |
USA |
|
|
Platform ID |
GPL17021 |
Series (2) |
GSE248206 |
Lineage-specific intolerance to oncogenic drivers restricts histological transformation [ATAC-Seq] |
GSE248207 |
Lineage-specific intolerance to oncogenic drivers restricts histological transformation |
|
Relations |
BioSample |
SAMN38320971 |
SRA |
SRX22573287 |