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| Status |
Public on Mar 31, 2018 |
| Title |
Human OCI-AML3 cells: Control vs. Atg5-shRNA or NPM1-shRNA inducibly expressed |
| Organism |
Homo sapiens |
| Experiment type |
Expression profiling by array
|
| Summary |
Transcriptional profiling of human OCI-AML3 cells stably expressing inducibly Atg5 shRNA or NPM1-shRNA
Goal was to determine the effects of knockdown of Atg5 or NPM1 on global ES gene expression of human Leukemia OCI-AML3 cells.
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| |
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| Overall design |
Control (Dox-) vs. inducible knock down of Atg5 (Dox+) or NPM1 (Dox+) by doxycycline. Biological replicates: 3 control replicates, Atg5-shRNA 2 replicates, NPM1-shRNA 3 replicates
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| |
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| Contributor(s) |
Ji X, huang M |
| Citation missing |
Has this study been published? Please login to update or notify GEO. |
| Submission date |
Mar 13, 2015 |
| Last update date |
Nov 27, 2018 |
| Contact name |
Min Huang |
| E-mail(s) |
minhuang@stanford.edu
|
| Organization name |
Stanford Cancer Institute
|
| Department |
Internal Medicine
|
| Lab |
Beverly Mitchell
|
| Street address |
265 Campus Drive
|
| City |
Stanford |
| State/province |
CA |
| ZIP/Postal code |
94305 |
| Country |
USA |
| |
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| Platforms (1) |
| GPL13607 |
Agilent-028004 SurePrint G3 Human GE 8x60K Microarray (Feature Number version) |
|
| Samples (8)
|
| GSM1634045 |
Control OCI-AML3 Cell without doxycycline Replicate 1 |
| GSM1634046 |
Control OCI-AML3 Cell without doxycycline Replicate 2 |
| GSM1634047 |
Control OCI-AML3 Cell without doxycycline Replicate 3 |
| GSM1634048 |
OCI-AML3 cells: Atg5-shRNA inducibly expressed by doxycycline for 6 days-replicate 1 |
| GSM1634049 |
OCI-AML3 cells: Atg5-shRNA inducibly expressed by doxycycline for 6 days-replicate 2 |
| GSM1634050 |
OCI-AML3 cells: NPM1-shRNA inducibly expressed by doxycycline for 6 days-replicate 1 |
| GSM1634051 |
OCI-AML3 cells: NPM1-shRNA inducibly expressed by doxycycline for 6 days-replicate 2 |
| GSM1634052 |
OCI-AML3 cells: NPM1-shRNA inducibly expressed by doxycycline for 6 days-replicate 3 |
|
| Relations |
| BioProject |
PRJNA278297 |
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