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Status |
Public on Apr 30, 2014 |
Title |
Significance of SALL4 as a drug resistant factor in lung cancer |
Organism |
Homo sapiens |
Experiment type |
Expression profiling by array
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Summary |
To examine the mechanism by which SALL4 regulates drug sensitivity, approximately 65,000 molecules on a microarray chip were analyzed using lung cancer cells transduced with a SALL4 vector (pCMV6-SALL4) or a control vector (pCMV6-Mock) .
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Overall design |
SALL4 mRNA and protein expression markedly increased after 24 hr of SALL4 vector transduction, unlike that in the cells transduced with the control vector; these cells were used for microarray analysis.
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Contributor(s) |
Yanagihara N, Kobayashi D, Kuribayashi K, Tanaka M, Hasegawa T, Watanabe N |
Citation missing |
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Submission date |
Apr 08, 2014 |
Last update date |
Nov 27, 2018 |
Contact name |
Nozomi Yanagihara |
E-mail(s) |
yanagihara.nozomi@sapmed.ac.jp
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Organization name |
Sapporo Medical University School of Medicine
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Department |
Department of Clinical Laboratory Medicine
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Street address |
South-1, West-16, Chuo-Ku
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City |
Sapporo |
ZIP/Postal code |
060-8543 |
Country |
Japan |
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Platforms (1) |
GPL13607 |
Agilent-028004 SurePrint G3 Human GE 8x60K Microarray (Feature Number version) |
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Samples (4)
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Relations |
BioProject |
PRJNA244027 |