|
Status |
Public on Dec 21, 2021 |
Title |
BCL11A promotes myeloid leukemogenesis by abrogating the transcriptional activity of PU.1 |
Organism |
Mus musculus |
Experiment type |
Genome binding/occupancy profiling by high throughput sequencing Expression profiling by array
|
Summary |
This SuperSeries is composed of the SubSeries listed below.
|
|
|
Overall design |
Refer to individual Series
|
|
|
Citation(s) |
34714913 |
|
Submission date |
Mar 30, 2020 |
Last update date |
Mar 22, 2022 |
Contact name |
Takuro Nakamura |
E-mail(s) |
takuro-ind@umin.net
|
Phone |
81-3-3570-0462
|
Organization name |
Japanese Foundation for Cancer Research
|
Department |
The Cancer Institute
|
Lab |
Carcinogenesis
|
Street address |
3-8-31 Ariake, Koto-ku
|
City |
Tokyo |
ZIP/Postal code |
135-8550 |
Country |
Japan |
|
|
Platforms (2) |
GPL11180 |
[HT_MG-430_PM] Affymetrix HT MG-430 PM Array Plate |
GPL16417 |
Illumina MiSeq (Mus musculus) |
|
Samples (16)
|
|
This SuperSeries is composed of the following SubSeries: |
GSE147787 |
BCL11A promotes myeloid leukemogenesis by abrogating the transcriptional activity of PU.1 [ChIP-seq] |
GSE147797 |
BCL11A promotes myeloid leukemogenesis by abrogating the transcriptional activity of PU.1 [array] |
|
Relations |
BioProject |
PRJNA616392 |