|
Status |
Public on Jun 19, 2020 |
Title |
Essential and Opposite Roles of ARID1A in Coordinating Human Cardiogenesis and Neurogenesis from Pluripotent Stem Cells |
Organism |
Homo sapiens |
Experiment type |
Genome binding/occupancy profiling by high throughput sequencing Expression profiling by high throughput sequencing
|
Summary |
This SuperSeries is composed of the SubSeries listed below.
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Overall design |
Refer to individual Series
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Citation(s) |
32646524 |
|
Submission date |
Oct 24, 2019 |
Last update date |
Jul 16, 2020 |
Contact name |
Lei Yang |
E-mail(s) |
lyang7@iu.edu
|
Phone |
(317) 278-5233
|
Organization name |
Indiana University School of Medicine
|
Department |
Department of Pediatrics
|
Street address |
1044 W. Walnut St.
|
City |
Indianapolis |
State/province |
Indiana |
ZIP/Postal code |
46202 |
Country |
USA |
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Platforms (2) |
GPL18573 |
Illumina NextSeq 500 (Homo sapiens) |
GPL24676 |
Illumina NovaSeq 6000 (Homo sapiens) |
|
Samples (8)
|
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This SuperSeries is composed of the following SubSeries: |
GSE139260 |
Genome-wide maps of ARID1A binding genes in H9 human embryonic stem cells. |
GSE139329 |
Assay for Transposase Accessible Chromatin with high-throughput sequencing (ATAC-seq) revealed genomic chromatin accessibilities change induced by loss of ARID1A in differentiated (day 4) H9 hESCs |
GSE139342 |
Single cell RNA-seq revealed different cell types induced by loss of ARID1A in undifferentiated and differentiation (day 10) H9 hESCs. |
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Relations |
BioProject |
PRJNA579345 |