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Series GSE126918 Query DataSets for GSE126918
Status Public on Mar 26, 2020
Title Loss of CHD1 promotes chromatin dysregulation leading to heterogeneous mechanisms of resistance to hormone therapy in prostate cancer
Organisms Homo sapiens; synthetic construct
Experiment type Expression profiling by high throughput sequencing
Genome binding/occupancy profiling by high throughput sequencing
Other
Summary This SuperSeries is composed of the SubSeries listed below.
 
Overall design Refer to individual Series
Web link https://pubmed.ncbi.nlm.nih.gov/32220301/
 
Citation(s) 32220301
Submission date Feb 22, 2019
Last update date Jun 25, 2020
Contact name Ping Mu
E-mail(s) muping817@gmail.com
Organization name UT Southwestern Medical Center
Department Human Oncology and Pathogenesis
Lab Mu Lab
Street address 6000 Harry Hines Blvd., NA6.308A
City Dallas
State/province TX
ZIP/Postal code 76092
Country USA
 
Platforms (3)
GPL16791 Illumina HiSeq 2500 (Homo sapiens)
GPL19604 Illumina HiSeq 2500 (synthetic construct)
GPL20301 Illumina HiSeq 4000 (Homo sapiens)
Samples (453)
GSM3630943 shCHD1-2_1_1 (RNA-seq)
GSM3630944 shCHD1-2_1_2 (RNA-seq)
GSM3630945 shCHD1-2_2_1 (RNA-seq)
This SuperSeries is composed of the following SubSeries:
GSE126917 Loss of CHD1 promotes chromatin dysregulation leading to heterogeneous mechanisms of resistance to hormone therapy in prostate cancer [RNA-seq]
GSE127241 Loss of CHD1 promotes chromatin dysregulation leading to heterogeneous mechanisms of resistance to hormone therapy in prostate cancer [ATAC-seq]
GSE127957 Loss of CHD1 promotes chromatin dysregulation leading to heterogeneous mechanisms of resistance to hormone therapy in prostate cancer [shRNA screen]
Relations
BioProject PRJNA523724

Download family Format
SOFT formatted family file(s) SOFTHelp
MINiML formatted family file(s) MINiMLHelp
Series Matrix File(s) TXTHelp

Supplementary file Size Download File type/resource
GSE126918_RAW.tar 1.9 Gb (http)(custom) TAR (of BW, TXT)
SRA Run SelectorHelp

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