BORIS/CTCFL epigenetically reprograms clustered CTCF binding sites into alternative transcriptional start sites.

NOTCH2 Promotes Osteoclast Maturation and Metabolism and Modulates the Transcriptome Profile During Osteoclastogenesis

Transcriptomic characterization of colon smooth muscle in young murine.

Red blood cells-heme-exposure and tumor-associated macrophage identity

Hemorrhage-induced NRF2 signaling in tumor-associated macrophages drives cancer growth, invasion, and immunotherapy resistance

Smooth muscle-derived adventitial progenitor cells direct atherosclerotic plaque composition complexity in a Klf4-dependent manner

Red blood cell heme drives the generation of TAM-like macrophages with an immunocompromized phenotype

Mixed cell-type spheroids endorse the generation and maintenance of heme-TAMs in an experimental tumor microenvironment

Heme-TAMs prevent cancer cell apoptosis and drive growth, invasiveness, and metastasis

Heme-TAMs are resistant to tumoricidal transformation by INFγ

Heme-signaling progresses via NRF2 to educate heme-TAM transformation, tumor cell growth, and invasiveness

Increased Enhancer—Promoter Interactions during Developmental Enhancer Activation in Mammals

Gene expression of immune cells in a lung cancer mouse model

Single cell RNA sequencing reveals endothelial cell killing and resolution pathways in experimental malaria-associated acute respiratory distress syndrome

RNA sequence analysis of mouse retina samples obtained from ApoE-/- mice infused with Ang II or saline

Microenvironmental CXCL12 Deletion Enhances Flt3-ITD Acute Myeloid Leukemia Stem Cell Response to Therapy by Reducing p38 MAPK Signaling

pH dependence of a GPR4 selective antagonist hampers its therapeutic potential

Heme exposure induces the phenotype of Hmox1high, Marcohigh, and MHC class IIlow macrophages

Sequencing of the hippocampal transcriptome of vestibular spatial disorientation mice

Keratin 5 Basal Cells are Temporally Regulated Developmental and Tissue Repair Progenitors in Bladder Urothelium.