Epigenetic reprogramming at estrogen-receptor binding sites alters the 3D chromatin landscape in endocrine resistant breast cancer [WGS]

Epigenetic reprogramming at estrogen-receptor binding sites alters 3D chromatin landscape in endocrine resistant breast cancer

Effect of overexpression of A3B and RNA editing calling in mouse tissues [WES]

Effect of overexpression of A3B and RNA editing calling in mouse tissues

Cancer cell genetics shaping of the tumor microenvironment reveals myeloid cell-centric exploitable vulnerabilities in hepatocellular carcinoma [WES]

Cancer cell genetics shaping of the tumor microenvironment reveals myeloid cell-centric exploitable vulnerabilities in hepatocellular carcinoma.

Cancer cell genetics shaping of the tumor microenvironmentreveals myeloid cell-centric exploitable vulnerabilities in hepatocellular carcinoma [WES 2]

Multiplexed functional genomic analysis of somatic 5’ untranslated region mutations across the spectrum of human prostate cancer

Multiplexed functional genomic analysis of somatic 5’ untranslated region mutations across the spectrum of human prostate cancer (Exome-Seq)

AKT/ERK-mediated activation of CDK7-MED1-AR axis drives resistance to pan BET bromodomain inhibitor ZEN-3694 in advanced prostate cancer [Exome-Seq]

Lens orientalis contributes quantitative trait loci and candidate genes associated with Ascochyta blight resistance in lentil

Whole exome sequencing of NT2.5 and NT2.5-LM cell lines

RNA-Seq, WGS, WXS of mouse myeloma tumors derived from Vk*MYC transgenic mice

Patient-derived organoid cultures model human primary liver cancer in vitro

Recombination initiation maps of individual human genomes

One-pot method for preparing DNA, RNA, and protein for multiomics analysis II

A maternal-effect Padi6 variant causes nuclear and cytoplasmic abnormalities in oocytes as well as failure of epigenetic reprogramming and zygotic genome activation in embryos [WGS]

A maternal-effect Padi6 variant causes nuclear and cytoplasmic abnormalities in oocytes as well 2 as failure of epigenetic reprogramming and zygotic genome activation in embryos

Physiologic expression of Srsf2(P95H) causes myeloid expansion, impaired competitive stem cell function and initiates the myeloproliferative/myelodysplastic syndrome in vivo [WES]

Physiologic expression of Srsf2(P95H) causes myeloid expansion, impaired competitive stem cell function and initiates the myeloproliferative/myelodysplastic syndrome in vivo.