Dynamics of gene silencing during X inactivation using allele-specific RNA-Seq

CTCF binding polarity determines chromatin looping [4C]

CTCF binding polarity determines chromatin looping

Cause and consequence of tethering a sub-TAD to different nuclear compartments

Dissection of a super-enhancer in vivo (ATAC-seq)

Genetic dissection of the α-globin super-enhancer

Polarized regulatory landscape and Wnt responsiveness underlie Hox activation in embryos

OmoMYC blunts promoter invasion by oncogenic MYC to inhibit gene expression characteristic of MYC-dependent tumors

Cdx and T Brachyury co-activate growth signaling in the embryonic axial progenitor niche.

Cdx is crucial for the timing mechanism driving collinear Hox activation and defines a trunk segment in the Hox cluster topology

Tissue-specific CTCF/Cohesin-mediated chromatin architecture delimits enhancer interactions and function in vivo

In situ CAPTURE of chromatin interactions by biotinylated dCas9

Enhancer invasion shapes MYCN dependent transcriptional amplification in neuroblastoma

YAP Partially Reprograms Chromatin Accessibility to Directly Induce Adult Cardiogenesis in Vivo

Mammalian ISWI and SWI/SNF selectively mediate binding of distinct groups of transcription factors

Simultaneous quantifications of protein-DNA contacts and transcriptomics in single cells

Simultaneous quantifications of protein-DNA contacts and transcriptomics in single cells (mESC data)

LncRNA-induced spread of Polycomb controlled by genome architecture, RNA abundance, and CpG island DNA

LncRNA-induced spread of Polycomb controlled by genome architecture, RNA abundance, and CpG island DNA [RIP-seq]

The ChAHP complex counteracts chromatin looping at Ctcf sites that have emerged from recent SINE expansions