Germline nuclear RNAi in C. elegans represses transgenerational inheritance of heat-induced gene transcriptional activation (chip-seq analysis)

Decoupling the downstream effects of germline nuclear RNAi reveals that transcriptional repression and heritable RNAi are independent of the H3K9me3 response in C. elegans [ChIP-seq]

Caenorhabditis elegans heterochromatin factor SET-32 plays an essential role in transgenerational initiation of nuclear RNAi-mediated epigenetic silencing (ChIP-Seq)

SET-9 and SET-26 are H3K4me3 readers and play critical roles in germline development and longevity

The transcriptional coactivator CBP/p300 is an evolutionarily conserved node that promotes longevity in response to mitochondrial stress [ChIP-seq]

The C. elegans ortholog of TDP-43 regulates the chromatin localization of the heterochromatin protein 1 homolog, HPL-2

To identify candidate genes whose expressions were regulated by the histone H4 modifications during P. aeruginosa PA01 exposure

Transcriptional suppression of sphingolipid catabolism controls pathogen resistance in C. elegans [ChIP-Seq]

Asymmetric Segregation of the Nucleosome Remodeling and Deacetylase Complex during Asymmetric Cell Divisions

Coordinated Maintenance of H3K36/K27 Methylation by Histone Demethylases Preserves Germ Cell Identity and Immortality

The nuclear hormone receptor NHR-86 controls anti-pathogen responses in C. elegans [ChIP-seq]

ChIP-Seq Files : The Hypoxia Response Pathway Promotes PEP Carboxykinase And Gluconeogenesis In C. elegans

The DREAM complex promotes gene body deposition of H2A.Z for target repression

C. elegans KLF-1 promoter binding sites

H3K4me3 involvement in long-term effects of sulfomethoxazole on Caenorhabditis elegans over 11 consecutive generations and their first non-exposed progeny