Deciphering Fatty Acid Biosynthesis-Driven Molecular Subtypes in Pancreatic Ductal Adenocarcinoma with Prognostic Insights

Nanostring analysis of neonatal brains following bacterial infection

Onocgenic cell marking and single-cell transcriptomics reveal cell type and time-resolved actions of von Hippel Lindau inactivation in the kidney

Lactic acid modifies gene expression and chromatin accessibility of bone marrow-derived macrophages during lipopolysaccharide stimulation (RNA-seq and ATAC-seq)

IMPDH2 filaments protect from neurodegeneration in AMPD2 deficiency

Synthetic reversed sequences reveal default genomic states

Synthetic reversed sequences reveal default genomic states [mESC RNAseq]

Synthetic reversed sequences reveal default genomic states [mESC_ChIP-Seq]

Synthetic reversed sequences reveal default genomic states [mESC_ATACseq]

Differentially expressed genes induced by UVB stimulation between the P2ry6+/+ and P2ry6-/- mice

Differentially expressed genes induced with or without UVB stimulation in mice

Gut complement induced by the microbiota combats pathogens and spares commensals

NF-kB Broadly Orchestrates Nucleosome Remodeling during the Primary Response to Toll-Like Receptor 4 Signaling [scATAC-seq]

Toll-Like Receptor-Induced Nucleosome Remodeling Achieved by Broadly Acting NF-kB in Collaboration with Transcription Factors Conferring Selectivity [BMDM_ATAC_2]

Dietary intervention reverses molecular signatures of hepatocellular senescence in the GAN diet-induced obese and biopsy-confirmed mouse model of NASH

Streamlined identification of clinically and functionally relevant genetic regulators of lower tract urogenital development

Safe and effective immunostimulatory therapy through vector-controlled cytokine delivery

Nutrient-derived signals regulate eosinophil adaptation to the small intestine

Toll-Like Receptor-Induced Nucleosome Remodeling Achieved by Broadly Acting NF-kB in Collaboration with Transcription Factors Conferring Selectivity [RNA-Seq]

Toll-Like Receptor-Induced Nucleosome Remodeling Achieved by Broadly Acting NF-kB in Collaboration with Transcription Factors Conferring Selectivity [ChIP-Seq]