Transcriptional Analysis of Wild Type and KLF2-/- Peritoneal Macrophages

Transcriptome analysis of mice dermal white adipose tissue during aging

Changes in gut microbiota induced by oral pathobiont deteriorate the pathology of non-alcoholic fatty liver disease

Hepatocyte-specific knockout of Nicotinamide phosphoribosyltransferase increases susceptibility for liver fibrosis in mice

p300 suppresses the transition of myelodysplastic syndrome to acute myeloid leukemia [RNA-Seq]

p300 suppresses the transition of myelodysplastic syndrome to acute myeloid leukemia [ATAC-Seq]

p300 suppresses the transition of myelodysplastic syndrome to acute myeloid leukemia [ChIP-seq]

p300 suppresses the transition of myelodysplastic syndrome to acute myeloid leukemia [DIP-seq]

p300 suppresses the transition of myelodysplastic syndrome to acute myeloid leukemia

An expansion of genomic regulatory complexity underlies vertebrate neuronal diversity

p300 suppresses the transition of myelodysplastic syndrome to acute myeloid leukemia [ChIP-seq II]

TGF-β/Nodal signaling boosts glucose uptake during mesendoderm differentiation by inducing a transmembrane micropeptide

In vitro generation of mature murine plasma cells uncovers adaptive Prdm1 expression

A Growth Factor-Expressing Macrophage subpopulation orchestrates regenerative inflammation via GDF-15 [scRNA-seq]

Transcriptional reprogramming of somatic cells after nuclear transfer to mouse 4-cell embryos

Integrative proteome analyses implicate aberrant RNA splicing impairs developmental potential of aged mouse oocytes

Methylomes of macrophage-DC progenitors (MDPs), monocytes, common monocyte progenitors (cMoPs), common dendritic cell progenitors (CDPs), plasmacytoid dendritic cells (pDCs), and cDC CD8a+ as well as cDC CD11b+ dendritic cells

Maladaptive functional changes in alveolar fibroblasts due to perinatal hyperoxia impair epithelial differentiation

Next generation sequencing of unstimulated and TNF-alpha-stimulated WT and DREAM-null neutrophils

Hepatocyte-specific perturbation of the NAD+ biosynthetic pathways in mice promotes reversible NASH-like phenotypes