Wapl repression by Pax5 promotes loop extrusion and V gene recombination at the Igh locus

Repression of the B cell identity factor Pax5 is not required for plasma cell development

Biallelic PAX5 mutations cause hypogammaglobulinemia,sensorimotor deficits and autism spectrum disorder

The Xbp1-regulated transcription factor Mist1 restricts antibody secretion by restraining Blimp1 expression in plasma cells

Stage-specific control of early B cell development by the transcription factor Ikaros

Multifunctional role of the transcription factor Blimp1 in coordinating plasma cell differentiation

Essential role of the transcription factors E2A and E2-2 in germinal center B cell and plasma cell development

Hobit and Blimp1 instruct a universal transcriptional program of tissue-residency in lymphocytes

CXCR5+ follicular cytotoxic T cells control viral infection in B cell follicles

Pax5 regulates B cell immunity by promoting PI3K signaling via PTEN downregulation

Precocious expression of Blimp1 in the B cell lineage causes autoimmunity

Ikaros prevents autoimmunity by promoting anergy induction and restraining Toll-like receptor signaling in B cells

The PAX5-JAK2 translocation acts as a dual-hit mutation that promotes aggressive B-cell leukemia via nuclear STAT5 activation

Enhancers of the PAIR4 regulatory module promote distal VH gene recombination at the Igh locus

Essential role of the Pax5 C-terminal domain in controlling B cell commitment and development

Transcriptional function of E2A, Ebf1, Pax5, Ikaros and Aiolos analyzed by in vivo acute protein degradation in early B cell development [ATAC-seq]

Transcriptional function of E2A, Ebf1, Pax5, Ikaros and Aiolos analyzed by in vivo acute protein degradation in early B cell development [CUT&RUN]

Transcriptional function of E2A, Ebf1, Pax5, Ikaros and Aiolos analyzed by in vivo acute protein degradation in early B cell development