Synergy between variant PRC1 complexes defines Polycomb-mediated gene repression (ChIP-Seq)

Synergy between variant PRC1 complexes defines Polycomb-mediated gene repression (RNA-Seq)

Synergy between variant PRC1 complexes defines Polycomb-mediated gene repression

Variant PRC2.1 and PRC2.2 co-operate to direct H3K27 methylations in ESCs [ChIP-seq]

Variant PRC2.1 and PRC2.2 co-operate to direct H3K27 methylations in ESCs

PRC2.1 and PRC2.2 synergize to co-ordinate H3K27 tri-methylation. 

CDK-Mediator and FBXL19 cooperate in the induction of developmental genes by promoting regulatory interactions [ChIP-seq]

CDK-Mediator and FBXL19 cooperate in the induction of developmental genes by promoting regulatory interactions

PRC1 drives Polycomb-mediated gene repression by controlling transcription initiation and burst frequency [ChIP-seq]

PRC1 drives Polycomb-mediated gene repression by controlling transcription initiation and burst frequency [RNA-seq]

PRC1 drives Polycomb-mediated gene repression by controlling transcription initiation and burst frequency

BAP1 constrains pervasive H2AK119ub1 to control the transcriptional potential of the genome [ChIP-seq]

BAP1 constrains pervasive H2AK119ub1 to control the transcriptional potential of the genome [ATAC-seq]

BAP1 constrains pervasive H2AK119ub1 to control the transcriptional potential of the genome [RNA-seq]

BAP1 constrains pervasive H2AK119ub1 to control the transcriptional potential of the genome.

H3K4 Methylation in β-cells prevents transcriptional downregulation and variance associated with type 2 diabetes [ChIP-seq]

H3K4 Methylation in β-cells prevents transcriptional downregulation and variance associated with type 2 diabetes [RNA-seq]

H3K4 Methylation in β-cells prevents transcriptional downregulation and variance associated with type 2 diabetes.

Distinct roles for CDK-Mediator in controlling Polycomb-dependent chromosomal interactions and priming genes for induction (ChIP-Seq)

Distinct roles for CDK-Mediator in controlling Polycomb-dependent chromosomal interactions and priming genes for induction (RNA-Seq)