Chemogenetic activation of astrocytes in the hippocampus and cortex changes the transcriptome of microglia

N6-methyladenosine modification is essential for prostate cancer cells

Interleukin-10/CSF1R fusion protein rejuvenates the exhausted T cells for executing antitumor activity

Interleukin-10/CSF1R fusion protein rejuvenates the exhausted T cells for executing antitumor activity II

Interleukin-10/CSF1R fusion protein rejuvenates the exhausted T cells for executing antitumor activity III

Interleukin-10/CSF1R fusion protein rejuvenates the exhausted T cells for executing antitumor activity IV

Interleukin-10/CSF1R fusion protein rejuvenates the exhausted T cells for executing antitumor activity V

Perturbed epigenetic transcriptional regulation in AML with IDH mutations causes increased susceptibility to NK cells [methylation]

Perturbed epigenetic transcriptional regulation in AML with IDH mutations causes increased susceptibility to NK cells [TT-Seq]

Perturbed epigenetic transcriptional regulation in AML with IDH mutations causes increased susceptibility to NK cells

Transcriptional network governing extraembryonic endoderm cell fate choice

Effect on gene expression profilng of BTSCs following HDAC1/2 inhibition with romidepsin.

Genomewide profiling of H3K27ac following HDAC1/2 inhibition with romidepsin and evaluation of co-regulated mechanisms by HDAC2 and SMAD3 in BTSC genome.

A CSF-1R-blocking antibody/IL-10 fusion protein increases anti-tumor immunity by effectuating tumor-resident CD8+ T cells

Concentration- and co-factor-dependent Runx DNA binding site choices during early T-development

Runx transcription factors regulate early T-developmental progression by inducing T-lineage identity and innate lymphoid cell program genes via gene network regulation

Transcriptomic changes in cervical cells during the menstrual cycle are evident but insufficient to predict window of implantation

A group of WRKY transcription factors form a protein complex with PHD-containing proteins to repress the transcription of stress responsive genes [RNA-Seq]

A group of WRKY transcription factors form a protein complex with PHD-containing proteins to repress the transcription of stress responsive genes [ChIP-Seq]

A group of WRKY transcription factors form a protein complex with PHD-containing proteins to repress the transcription of stress responsive genes