A TCF1-driven regulatory network determines homeostatic versus pathogenic state of Th17 cells [RNA-seq]

A TCF1-driven regulatory network determines homeostatic versus pathogenic state of Th17 cells

Effect of macrophage SNO-Septin2 in aortic aneurysm development

Effect of SNO-Septin2 in macrophage inflammation and migration

CEACAM1-engineered MSCs have a broad spectrum of immunomodulatory functions and therapeutic potential via cell-to-cell interaction

The effect of lnc-ORA knockout on muscle fiber type transformation in mouse leg muscle.

Translational reprogramming during epithelial-mesenchymal transition is associated with fine-tuning of ribosome composition

Transcriptomic analysis of gastric cancer cells in response to stable over-expression of circRNA derived from hnRNPU (circ-hnRNPU)

Dynamic coupling of R-loops with transcriptional pausing modulates tissue development in Drosophila

Gene expression-based, individualized outcome prediction for surgically treated pancreatic cancer patients

HeLa input control in high salt condition

Oxytocin Signaling Regulates the Homeostatic Response to Cold Stress in Ectothermic Vertebrate

RNAseq of mRNA from whole mouse liver

Identifying the molecular mechanisms that underlie impaired kidney development due to maternal diabetes.

Next generation sequencing for quantitative analysis of double-negative (DN) thymocyte transcriptomes from Toxf/f and Toxf/fCd4Cre mice.

AMTB, a TRPM8 Antagonist, Suppresses Growth and Metastasis of Osteosarcoma through Repressing the TGFβ Signaling Pathway

Time-resolved and multi-tissue RNAseq provided new insights on the immune responses of European eels following infection with Aeromonas hydrophila

Integrated Profiling and Immunohistochemistry Analyses Link circ-BRIP1, circ-EPHB4 and Their Host Genes with Imatinib Resistance in Gastrointestinal Stromal Tumors

Expression data of multple drug resistant bladder cancer cell model constructed by repeated exposure to pirarubicin treatment

Enterococcus faecalis-derived lactate mediated suppression of NF-κB and ERK pathways in macrophages to promote polymicrobial wound infection