| EVI5 is an oncogene that regulates the proliferation and metastasis of NSCLC cells. | EVI5 is an oncogene that regulates the proliferation and metastasis of NSCLC cells.
Cai T, Zhou J, Zeng Y, Du W, Zhang Y, Liu T, Fu Y, Huang JA, Qian Q, Zhu J, Ling C, Liu Z., Free PMC Article | 02/6/2021 |
| The allele and genotype frequencies of rs6680578 and rs11810217 were not significantly different between relapsing-remitting multiple sclerosis (MS) cases and controls. However, in the rs10735781 the GG genotype was significantly associated with MS risk in recessive and co-dominant models. T G T haplotype (rs6680578, rs10735781 and rs11810217 respectively) was associated with MS risk. | Ecotropic Viral Integration Site 5 (EVI5) variants are associated with multiple sclerosis in Iranian population.
Mazdeh M, Ghafouri-Fard S, Noroozi R, Sayad A, Khani M, Taheri M, Davood Omrani M. | 07/7/2018 |
| EVI5 is an useful independent prognostic marker of survival and recurrence in hepatocellular carcinoma | EVI5 is a novel independent prognostic predictor in hepatocellular carcinoma after radical hepatectomy.
Tang J, Ou J, Xu C, Yi C, Xue F, Xu L, Lai F, Tang J, Li S, Kang T, Ding W, Wang B. | 05/12/2018 |
| EVI5 as a strong candidate disease risk gene in the 1p22.1 multiple sclerosis risk locus | A non-synonymous single-nucleotide polymorphism associated with multiple sclerosis risk affects the EVI5 interactome.
Didonna A, Isobe N, Caillier SJ, Li KH, Burlingame AL, Hauser SL, Baranzini SE, Patsopoulos NA, Oksenberg JR., Free PMC Article | 08/27/2016 |
| Data show that heat shock transcription factor 1/miR-135b/reversion-inducing-cysteine-rich protein with kazal motifs and ecotropic viral integration site 5 axis provides novel insight into the mechanisms of hepatocellular carcinoma metastasis. | MicroRNA-135b, a HSF1 target, promotes tumor invasion and metastasis by regulating RECK and EVI5 in hepatocellular carcinoma.
Li Y, Xu D, Bao C, Zhang Y, Chen D, Zhao F, Ding J, Liang L, Wang Q, Liu L, Li J, Yao M, Huang S, He X., Free PMC Article | 01/16/2016 |
| in HLA-DRB1 positive patients, EVI5 was associated with attacks of greater severity and worse recovery in Multiple sclerosis . | Multiple sclerosis susceptibility genes: associations with relapse severity and recovery.
Mowry EM, Carey RF, Blasco MR, Pelletier J, Duquette P, Villoslada P, Malikova I, Roger E, Kinkel RP, McDonald J, Bacchetti P, Waubant E., Free PMC Article | 06/7/2014 |
| cellular functions exhibited by the Evi5 family members | The Evi5 family in cellular physiology and pathology.
Lim YS, Tang BL. | 08/31/2013 |
| An analysis and fine mapping of GFI-EVI5-RPL5-FAM69A locus, genotyping eight Tag-single nucleotide polymorphisms in 732 multiple sclerosis patients and 974 controls from Spain, was performed. | Tag-SNP analysis of the GFI1-EVI5-RPL5-FAM69 risk locus for multiple sclerosis.
Alcina A, Fernández O, Gonzalez JR, Catalá-Rabasa A, Fedetz M, Ndagire D, Leyva L, Guerrero M, Arnal C, Delgado C, Lucas M, Izquierdo G, Matesanz F, Alcina A, Fernández O, Gonzalez JR, Catalá-Rabasa A, Fedetz M, Ndagire D, Leyva L, Guerrero M, Arnal C, Delgado C, Lucas M, Izquierdo G, Matesanz F., Free PMC Articles: PMC2987353, PMC2987353 | 09/20/2010 |
| Stem cell exhaustion due to Runx1 deficiency is prevented by Evi5 activation in leukemogenesis. | Stem cell exhaustion due to Runx1 deficiency is prevented by Evi5 activation in leukemogenesis.
Jacob B, Osato M, Yamashita N, Wang CQ, Taniuchi I, Littman DR, Asou N, Ito Y., Free PMC Article | 03/29/2010 |
| Observational study and meta-analysis of gene-disease association. (HuGE Navigator) | Replication of CD58 and CLEC16A as genome-wide significant risk genes for multiple sclerosis.
Hoppenbrouwers IA, Aulchenko YS, Janssens AC, Ramagopalan SV, Broer L, Kayser M, Ebers GC, Oostra BA, van Duijn CM, Hintzen RQ. | 12/2/2009 |
| EVI5 is a risk gene for multiple sclerosis. | EVI5 is a risk gene for multiple sclerosis.
Hoppenbrouwers IA, Aulchenko YS, Ebers GC, Ramagopalan SV, Oostra BA, van Duijn CM, Hintzen RQ, Hoppenbrouwers IA, Aulchenko YS, Ebers GC, Ramagopalan SV, Oostra BA, van Duijn CM, Hintzen RQ. | 01/21/2010 |
| Observational study of gene-disease association. (HuGE Navigator) | See all PubMed (4) articlesTag-SNP analysis of the GFI1-EVI5-RPL5-FAM69 risk locus for multiple sclerosis.
Alcina A, Fernández O, Gonzalez JR, Catalá-Rabasa A, Fedetz M, Ndagire D, Leyva L, Guerrero M, Arnal C, Delgado C, Lucas M, Izquierdo G, Matesanz F, Alcina A, Fernández O, Gonzalez JR, Catalá-Rabasa A, Fedetz M, Ndagire D, Leyva L, Guerrero M, Arnal C, Delgado C, Lucas M, Izquierdo G, Matesanz F. Multiple sclerosis susceptibility alleles in African Americans.
Johnson BA, Wang J, Taylor EM, Caillier SJ, Herbert J, Khan OA, Cross AH, De Jager PL, Gourraud PA, Cree BC, Hauser SL, Oksenberg JR. Risk alleles for multiple sclerosis in multiplex families.
D'Netto MJ, Ward H, Morrison KM, Ramagopalan SV, Dyment DA, DeLuca GC, Handunnetthi L, Sadovnick AD, Ebers GC. EVI5 is a risk gene for multiple sclerosis.
Hoppenbrouwers IA, Aulchenko YS, Ebers GC, Ramagopalan SV, Oostra BA, van Duijn CM, Hintzen RQ, Hoppenbrouwers IA, Aulchenko YS, Ebers GC, Ramagopalan SV, Oostra BA, van Duijn CM, Hintzen RQ. | 04/23/2008 |
| a novel centrosomal protein with a complex expression pattern and subcellular localization, possibly involved in centrosome stability and dynamics | EVI5 is a novel centrosomal protein that binds to alpha- and gamma-tubulin.
Faitar SL, Dabbeekeh JT, Ranalli TA, Cowell JK. | 01/21/2010 |
| Data provide evidence that EVI5 is an essential component of the protein machinery facilitating the final stages of cell septation at the end of mitosis. | EVI5 protein associates with the INCENP-aurora B kinase-survivin chromosomal passenger complex and is involved in the completion of cytokinesis.
Faitar SL, Sossey-Alaoui K, Ranalli TA, Cowell JK. | 01/21/2010 |
| Evi5 is a Rab11 binding protein and that Evi5 may cooperate with Rab11 to coordinate vesicular trafficking, cytokinesis, and cell cycle control independent of GTPase-activating protein function | Identification of Rab11 as a small GTPase binding protein for the Evi5 oncogene.
Westlake CJ, Junutula JR, Simon GC, Pilli M, Prekeris R, Scheller RH, Jackson PK, Eldridge AG., Free PMC Article | 01/21/2010 |