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    PROS1 protein S [ Homo sapiens (human) ]

    Gene ID: 5627, updated on 27-Aug-2024

    GeneRIFs: Gene References Into Functions

    GeneRIFPubMed TitleDate
    Duodenal mucosa of untreated celiac disease patients has altered expression of the GAS6 and PROS1 and the negative regulator tyrosine kinase TAM receptors subfamily.

    Duodenal mucosa of untreated celiac disease patients has altered expression of the GAS6 and PROS1 and the negative regulator tyrosine kinase TAM receptors subfamily.
    Perez F, Iribarren ML, Olexen CM, Ruera CN, Errasti AE, Guzman L, Garbi L, Carrera Silva EA, Chirdo FG.

    08/22/2024
    PROS1 is a crucial gene in the macrophage efferocytosis of diabetic foot ulcers: a concerted analytical approach through the prisms of computer analysis.

    PROS1 is a crucial gene in the macrophage efferocytosis of diabetic foot ulcers: a concerted analytical approach through the prisms of computer analysis.
    Shi H, Zhang Z, Yuan X, Liu G, Fan W, Wang W., Free PMC Article

    08/2/2024
    The Protein S Erlangen Mutation PROS1c.1904T>C (F635S) Suppresses Secretion.

    The Protein S Erlangen Mutation PROS1c.1904T>C (F635S) Suppresses Secretion.
    Reißig J, Cunningham S, Wandersee A, Brox R, Achenbach S, Strobel J, Hackstein H, Schneider S.

    08/1/2024
    Replication Study of the Association of GAS6 and PROS1 Polymorphisms with Behcet's Disease in a Japanese Population.

    Replication Study of the Association of GAS6 and PROS1 Polymorphisms with Behçet's Disease in a Japanese Population.
    Teshigawara T, Meguro A, Takeuchi M, Ishido M, Soejima Y, Hirahara L, Kirino Y, Ohno S, Mizuki N.

    06/24/2024
    Analysis of PROS1 mutations and clinical characteristics in three Chinese families with hereditary protein S deficiency.

    Analysis of PROS1 mutations and clinical characteristics in three Chinese families with hereditary protein S deficiency.
    Xu F, Zhou X, Jin Y, Yang L, Pan J, Wang M, Chen X.

    02/1/2024
    Activated protein C, protein S, and tissue factor pathway inhibitor cooperate to inhibit thrombin activation.

    Activated protein C, protein S, and tissue factor pathway inhibitor cooperate to inhibit thrombin activation.
    Li X, Song X, Mahmood DFD, Sim MMS, Bidarian SJ, Wood JP.,

    10/10/2023
    Protein C and S levels in patients with Thalassemia intermedia.

    Protein C and S levels in patients with Thalassemia intermedia.
    Mohammed NS., Free PMC Article

    01/14/2023
    Dysregulation of Protein S in COVID-19.

    Dysregulation of Protein S in COVID-19.
    Sim MMS, Wood JP., Free PMC Article

    12/31/2022
    AXL, along with PROS1, is overexpressed in papillary thyroid carcinoma and regulates its biological behaviour.

    AXL, along with PROS1, is overexpressed in papillary thyroid carcinoma and regulates its biological behaviour.
    Wei M, Wang Y, Liu Y, Li D, He X., Free PMC Article

    10/15/2022
    LncRNA RP3-525N10.2-NFKB1-PROS1 triplet-mediated low PROS1 expression is an onco-immunological biomarker in low-grade gliomas: a pan-cancer analysis with experimental verification.

    LncRNA RP3-525N10.2-NFKB1-PROS1 triplet-mediated low PROS1 expression is an onco-immunological biomarker in low-grade gliomas: a pan-cancer analysis with experimental verification.
    Zhou Y, Xiao D, Jiang X., Free PMC Article

    07/30/2022
    Protein S Erlangen: a novel PROS1 gene mutation associated with quantitative protein S deficiency.

    Protein S Erlangen: a novel PROS1 gene mutation associated with quantitative protein S deficiency.
    Schneider S, Reißig J, Weisbach V, Achenbach S, Strobel J, Hackstein H.

    06/11/2022
    Protein S-sulfhydration: Unraveling the prospective of hydrogen sulfide in the brain, vasculature and neurological manifestations.

    Protein S-sulfhydration: Unraveling the prospective of hydrogen sulfide in the brain, vasculature and neurological manifestations.
    Gupta R, Sahu M, Tripathi R, Ambasta RK, Kumar P.

    05/7/2022
    Protein S-Leu17Pro disrupts the hydrophobicity of its signal peptide causing a proteasome-dependent degradation.

    Protein S-Leu17Pro disrupts the hydrophobicity of its signal peptide causing a proteasome-dependent degradation.
    Okada K, Tamura S, Suzuki N, Odaira K, Mukaide M, Fujii W, Katsuragi Y, Suzuki A, Kanematsu T, Okamoto S, Suzuki N, Katsumi A, Matsushita T, Kojima T, Hayakawa F.

    04/2/2022
    S-palmitoylation of NOD2 controls its localization to the plasma membrane.

    S-palmitoylation of NOD2 controls its localization to the plasma membrane.
    Dixon CL, Fairn GD., Free PMC Article

    03/26/2022
    [Clinical manifestations and gene analysis of 18 cases of hereditary protein S deficiency].

    [Clinical manifestations and gene analysis of 18 cases of hereditary protein S deficiency].
    Zhang DL, Xue F, Fu RF, Chen YF, Liu XF, Liu W, Jia YJ, Li HY, Wang YH, Xiao ZJ, Zhang L, Yang RC., Free PMC Article

    03/5/2022
    A Sweet H2S/H2O2 Dual Release System and Specific Protein S-Persulfidation Mediated by Thioglucose/Glucose Oxidase.

    A Sweet H(2)S/H(2)O(2) Dual Release System and Specific Protein S-Persulfidation Mediated by Thioglucose/Glucose Oxidase.
    Ni X, Li X, Shen TL, Qian WJ, Xian M.

    02/12/2022
    Increased expression of Protein S in eyes with diabetic retinopathy and diabetic macular edema.

    Increased expression of Protein S in eyes with diabetic retinopathy and diabetic macular edema.
    Sugimoto M, Kondo M, Yasuma T, D'Alessandro-Gabazza CN, Toda M, Imai H, Nakamura M, Gabazza EC., Free PMC Article

    11/6/2021
    Artesunate and Tetramethylpyrazine Exert Effects on Experimental Cerebral Malaria in a Mechanism of Protein S-Nitrosylation.

    Artesunate and Tetramethylpyrazine Exert Effects on Experimental Cerebral Malaria in a Mechanism of Protein S-Nitrosylation.
    Zheng Z, Liu H, Wang X, Zhang Y, Qu S, Yang Y, Deng S, Chen L, Zhu X, Li Y.

    11/6/2021
    Five new mutations in the PROS1 gene associated with protein S deficiency in Polish patients screened for thrombophilia: efficacy of direct oral anticoagulant treatment.

    Five new mutations in the PROS1 gene associated with protein S deficiency in Polish patients screened for thrombophilia: efficacy of direct oral anticoagulant treatment.
    Wypasek E, Potaczek DP, Klajmon A, Zúñiga A, Undas A.

    10/23/2021
    Modulation of protein S and growth arrest specific 6 protein signaling inhibits pancreatic cancer cell survival and proliferation.

    Modulation of protein S and growth arrest specific 6 protein signaling inhibits pancreatic cancer cell survival and proliferation.
    Pilli VS, Datta A, Dorsey A, Liu B, Majumder R., Free PMC Article

    07/10/2021
    Recurrent PROC and novel PROS1 mutations in Vietnamese patients diagnosed with idiopathic deep venous thrombosis.

    Recurrent PROC and novel PROS1 mutations in Vietnamese patients diagnosed with idiopathic deep venous thrombosis.
    Do MD, Pham DV, Le LP, Gia Le LH, Minh Tran LB, Dang Huynh MD, Do QM, Vu HA, Nguyen NH, Mai TP.

    06/12/2021
    Protein S protects against allergic bronchial asthma by modulating Th1/Th2 balance.

    Protein S protects against allergic bronchial asthma by modulating Th1/Th2 balance.
    Asayama K, Kobayashi T, D'Alessandro-Gabazza CN, Toda M, Yasuma T, Fujimoto H, Okano T, Saiki H, Takeshita A, Fujiwara K, Fridman D'Alessandro V, Nishihama K, Totoki T, Inoue R, Takei Y, Gabazza EC.

    06/12/2021
    [Genetic and Clinical Characteristics of A Family with Combined PROC and PROS1 Genetic Variants].

    [Genetic and Clinical Characteristics of A Family with Combined PROC and PROS1 Genetic Variants].
    Wang X, Sheng GY, Zhang W, Zhao YX, Xia LJ, Jiang M.

    04/13/2021
    Two Novel Variants in the Protein S Gene PROS1 Are Associated with Protein S Deficiency and Thrombophilia.

    Two Novel Variants in the Protein S Gene PROS1 Are Associated with Protein S Deficiency and Thrombophilia.
    Juhl D, Kuta P, Shneyder M, Wünsche F, Nowak-Göttl U.

    04/3/2021
    Comparative study of hypercoagulability change in steady state and during vaso-occlusive crisis among Sudanese patients living with sickle cell disease.

    Comparative study of hypercoagulability change in steady state and during vaso-occlusive crisis among Sudanese patients living with sickle cell disease.
    Mohamed EA, Elgari MM, Babker AM, Waggiallah HA., Free PMC Article

    02/27/2021
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