| Expression of DNAJB9 and some other genes is more sensitive to SWCNTs in normal human astrocytes than glioblastoma cells. | Expression of DNAJB9 and some other genes is more sensitive to SWCNTs in normal human astrocytes than glioblastoma cells.
Minchenko DO, Rudnytska OV, Khita OO, Kulish YV, Viletska YM, Halkin OV, Danilovskyi SV, Ratushna OO, Minchenko OH. | 08/16/2023 |
| DNAJB9 suppresses the metastasis of triple-negative breast cancer by promoting FBXO45-mediated degradation of ZEB1. | DNAJB9 suppresses the metastasis of triple-negative breast cancer by promoting FBXO45-mediated degradation of ZEB1.
Kim HY, Kim YM, Hong S., Free PMC Article | 10/16/2021 |
| DNAJB9-positive monotypic fibrillary glomerulonephritis is not associated with monoclonal gammopathy in the vast majority of patients. | DNAJB9-positive monotypic fibrillary glomerulonephritis is not associated with monoclonal gammopathy in the vast majority of patients.
Said SM, Leung N, Alexander MP, Cornell LD, Fidler ME, Grande JP, Herrera LH, Sethi S, Zhang P, Nasr SH. | 08/7/2021 |
| Clinicopathological characteristics and outcome of patients with fibrillary glomerulonephritis: DNAJB9 is a valuable histologic marker. | Clinicopathological characteristics and outcome of patients with fibrillary glomerulonephritis: DNAJB9 is a valuable histologic marker.
Liang S, Chen D, Liang D, Xu F, Zhang M, Yang F, Zhu X, Li P, Zeng C. | 07/24/2021 |
| Hepatic DNAJB9 Drives Anabolic Biasing to Reduce Steatosis and Obesity. | Hepatic DNAJB9 Drives Anabolic Biasing to Reduce Steatosis and Obesity.
Sun F, Liao Y, Qu X, Xiao X, Hou S, Chen Z, Huang H, Li P, Fu S. | 03/13/2021 |
| Long non-coding RNA SNHG5 regulates chemotherapy resistance through the miR-32/DNAJB9 axis in acute myeloid leukemia. | Long non-coding RNA SNHG5 regulates chemotherapy resistance through the miR-32/DNAJB9 axis in acute myeloid leukemia.
Wang D, Zeng T, Lin Z, Yan L, Wang F, Tang L, Wang L, Tang D, Chen P, Yang M. | 11/21/2020 |
| New developments in the diagnosis of fibrillary glomerulonephritis. | New developments in the diagnosis of fibrillary glomerulonephritis.
Nasr SH, Fogo AB. | 10/24/2020 |
| Targeting DNAJB9, a novel ER luminal co-chaperone, to rescue DeltaF508-CFTR. | Targeting DNAJB9, a novel ER luminal co-chaperone, to rescue ΔF508-CFTR.
Huang Y, Arora K, Mun KS, Yang F, Moon C, Yarlagadda S, Jegga A, Weaver T, Naren AP., Free PMC Article | 10/24/2020 |
| these observations support a competition model whereby waning ER stress passively partitions ERdj4 and BiP to IRE1(LD) to initiate active repression of UPR signalling. | Unstructured regions in IRE1α specify BiP-mediated destabilisation of the luminal domain dimer and repression of the UPR.
Amin-Wetzel N, Neidhardt L, Yan Y, Mayer MP, Ron D., Free PMC Article | 06/13/2020 |
| Study detected a 4-fold higher abundance of serum DNAJB9 in fibrillary glomerulonephritis (FGN) patients when compared to controls, including patients with other glomerular diseases. Serum DNAJB9 levels were also negatively associated with estimated glomerular filtration rate in patients with FGN. | Serum levels of DNAJB9 are elevated in fibrillary glomerulonephritis patients.
Nasr SH, Dasari S, Lieske JC, Benson LM, Vanderboom PM, Holtz-Heppelmann CJ, Giesen CD, Snyder MR, Erickson SB, Fervenza FC, Leung N, Kurtin PJ, Alexander MP. | 04/4/2020 |
| Study reports that the endoplasmic reticulum luminal co-chaperone ERdj4/DNAJB9 is a selective IRE1 repressor that promotes a complex between the luminal Hsp70 BiP and the luminal stress-sensing domain of IRE1alpha. | A J-Protein Co-chaperone Recruits BiP to Monomerize IRE1 and Repress the Unfolded Protein Response.
Amin-Wetzel N, Saunders RA, Kamphuis MJ, Rato C, Preissler S, Harding HP, Ron D., Free PMC Article | 12/30/2017 |
| these results demonstrate that DNAJB9 is a downstream target of p53 that belongs to the group of negative feedback regulators of p53. | Genotoxic stress/p53-induced DNAJB9 inhibits the pro-apoptotic function of p53.
Lee HJ, Kim JM, Kim KH, Heo JI, Kwak SJ, Han JA., Free PMC Article | 08/1/2015 |
| ERdj4 and ERdj5 promote turnover of misfolded SP-C and this activity is dependent on their ability to stimulate BiP ATPase activity. | ERdj4 and ERdj5 are required for endoplasmic reticulum-associated protein degradation of misfolded surfactant protein C.
Dong M, Bridges JP, Apsley K, Xu Y, Weaver TE., Free PMC Article | 01/21/2010 |