| Functional evaluation of various ICAM3 transcript variants in diffuse large B-Cell lymphoma. | Functional evaluation of various ICAM3 transcript variants in diffuse large B-Cell lymphoma.
Li L, Yang C, Aruna, Zhou Q, Jiang X, Du W, Liu C, Lv P, Wang X, Fan G, Zhao S, Zhang X, Jin A, Shen W. | 12/10/2022 |
| Association of Circulating ICAM3 Concentrations with Severity and Short-term Outcomes of Acute Ischemic Stroke. | Association of Circulating ICAM3 Concentrations with Severity and Short-term Outcomes of Acute Ischemic Stroke.
Zicheng H, Xiao Y, Rongzhong H, Yongyong L, Haitao R, Tingting S. | 12/18/2021 |
| Changes in the Surface Expression of Intercellular Adhesion Molecule 3, the Induction of Apoptosis, and the Inhibition of Cell-Cycle Progression of Human Multidrug-Resistant Jurkat/A4 Cells Exposed to a Random Positioning Machine. | Changes in the Surface Expression of Intercellular Adhesion Molecule 3, the Induction of Apoptosis, and the Inhibition of Cell-Cycle Progression of Human Multidrug-Resistant Jurkat/A4 Cells Exposed to a Random Positioning Machine.
Sokolovskaya A, Korneeva E, Zaichenko D, Virus E, Kolesov D, Moskovtsev A, Kubatiev A., Free PMC Article | 11/21/2020 |
| Vascular injury biomarkers and stroke risk: A population-based study. | Vascular injury biomarkers and stroke risk: A population-based study.
Pletsch-Borba L, Grafetstätter M, Hüsing A, Johnson T, González Maldonado S, Groß ML, Kloss M, Hoffmeister M, Bugert P, Kaaks R, Kühn T. | 09/12/2020 |
| we identify a potential CSC regulator and suggest a novel mechanism by which ICAM3 governs cancer cell stemness and inflammation. | ICAM3 mediates inflammatory signaling to promote cancer cell stemness.
Shen W, Xie J, Zhao S, Du R, Luo X, He H, Jiang S, Hao N, Chen C, Guo C, Liu Y, Chen Y, Sun P, Yang S, Luo N, Xiang R, Luo Y., Free PMC Article | 05/25/2019 |
| Lewis-antigen-containing ICAM-2/3 on Jurkat leukemia cells interact with DC-SIGN to regulate DC functions. | Lewis-antigen-containing ICAM-2/3 on Jurkat leukemia cells interact with DC-SIGN to regulate DC functions.
Jin X, Bu Q, Zou Y, Feng Y, Wei M. | 12/22/2018 |
| exploration of the underlying mechanism demonstrated that ICAM3 not only binds to LFA-1 with its extracellular domain and structure protein ERM but also to lamellipodia with its intracellular domain which causes a tension that pulls cells apart (metastasis). | ICAM3 mediates tumor metastasis via a LFA-1-ICAM3-ERM dependent manner.
Shen W, Zhang X, Du R, Fan Y, Luo D, Bao Y, Yang W, Luo N, Luo Y, Zhao S. | 08/11/2018 |
| Increased expression of PECAM-1, ICAM-3, and VCAM-1 in colonic biopsies from patients with inflammatory bowel disease (IBD) in clinical remission is associated with subsequent flares; this suggests that increases in the expression of these proteins may be early events that lead to flares in patients with IBD | Increased Cell Adhesion Molecules, PECAM-1, ICAM-3, or VCAM-1, Predict Increased Risk for Flare in Patients With Quiescent Inflammatory Bowel Disease.
Gu P, Theiss A, Han J, Feagins LA., Free PMC Article | 03/24/2018 |
| with larger patient groups and preferably detailed histopathological and clinical evaluations, are needed to explain the severity of ICAM-1, ICAM-2, and ICAM-3 molecules in Barrett's esophagus | Investigation of intercellular adhesion molecules (ICAMs) gene expressions in patients with Barrett's esophagus.
Alasehirli B, Oğuz E, Oksuzler E, Koruk I, Oztuzcu S, Ozkara E, Karakok M, Erbagcı AB, Demiryurek AT. | 06/28/2014 |
| ICAM3 acts as recognition receptors in the phagocytosis portals of macrophages for engulfment of apoptotic neutrophils. | Novel role of ICAM3 and LFA-1 in the clearance of apoptotic neutrophils by human macrophages.
Kristóf E, Zahuczky G, Katona K, Doró Z, Nagy É, Fésüs L. | 03/29/2014 |
| this data clearly indicate that ICAM-3 promotes drug resistance via inhibition of apoptosis. | ICAM-3 endows anticancer drug resistance against microtubule-damaging agents via activation of the ICAM-3-AKT/ERK-CREB-2 pathway and blockage of apoptosis.
Ahn KC, Choi JY, Kim JS, Hwang SG, Kim WJ, Park JK, Um HD. | 01/18/2014 |
| Intercellular adhesion molecule (ICAM)-3 mRNA is upregulated in non-adherent endothelial forming cells. | Characterization of a distinct population of circulating human non-adherent endothelial forming cells and their recruitment via intercellular adhesion molecule-3.
Appleby SL, Cockshell MP, Pippal JB, Thompson EJ, Barrett JM, Tooley K, Sen S, Sun WY, Grose R, Nicholson I, Levina V, Cooke I, Talbo G, Lopez AF, Bonder CS., Free PMC Article | 04/27/2013 |
| Results indicate that the ICAM-3 gene promoter is negatively regulated by RUNX3. | RUNX3 regulates intercellular adhesion molecule 3 (ICAM-3) expression during macrophage differentiation and monocyte extravasation.
Estecha A, Aguilera-Montilla N, Sánchez-Mateos P, Puig-Kröger A., Free PMC Article | 09/1/2012 |
| analysis of activated apoptotic cells induce dendritic cell maturation via engagement of Toll-like receptor 4 (TLR4), dendritic cell-specific intercellular adhesion molecule 3 (ICAM-3)-grabbing nonintegrin (DC-SIGN), and beta2 integrins | Activated apoptotic cells induce dendritic cell maturation via engagement of Toll-like receptor 4 (TLR4), dendritic cell-specific intercellular adhesion molecule 3 (ICAM-3)-grabbing nonintegrin (DC-SIGN), and β2 integrins.
Pathak SK, Sköld AE, Mohanram V, Persson C, Johansson U, Spetz AL., Free PMC Article | 07/21/2012 |
| ICAM-3 may be an important adhesion molecule involved in chemotaxis to apoptotic human leukocytes. | Apoptotic cell-derived ICAM-3 promotes both macrophage chemoattraction to and tethering of apoptotic cells.
Torr EE, Gardner DH, Thomas L, Goodall DM, Bielemeier A, Willetts R, Griffiths HR, Marshall LJ, Devitt A., Free PMC Article | 07/7/2012 |
| the molecular basis of allergen-induced Th2 cell polarization | Retagging identifies dendritic cell-specific intercellular adhesion molecule-3 (ICAM3)-grabbing non-integrin (DC-SIGN) protein as a novel receptor for a major allergen from house dust mite.
Emara M, Royer PJ, Mahdavi J, Shakib F, Ghaemmaghami AM., Free PMC Article | 04/14/2012 |
| the cross-talk between neutrophils and NK cells is mediated by ICAM-3 and CD11d/CD18, respectively. | On the potential involvement of CD11d in co-stimulating the production of interferon-γ by natural killer cells upon interaction with neutrophils via intercellular adhesion molecule-3.
Costantini C, Micheletti A, Calzetti F, Perbellini O, Tamassia N, Albanesi C, Vermi W, Cassatella MA., Free PMC Article | 02/11/2012 |
| Single nucleotide polymorphisms in ICAM3 gene is associated with lymphoma. | Polymorphisms in integrin genes and lymphoma risk.
Shen M, Zheng T, Lan Q, Zhang Y, Hosgood HD 3rd, Zahm SH, Holford TR, Leaderer B, Yeager M, Yuenger J, Chanock S, Rothman N., Free PMC Article | 08/20/2011 |
| induction of morphological polarization in primary T lymphocytes and Jurkat cells enhances Kidins220/ARMS colocalization with ICAM-3 | The neuronal protein Kidins220/ARMS associates with ICAM-3 and other uropod components and regulates T-cell motility.
Jean-Mairet RM, López-Menéndez C, Sánchez-Ruiloba L, Sacristán S, Rodríguez-Martínez M, Riol-Blanco L, Sánchez-Mateos P, Sánchez-Madrid F, Rodríguez-Fernández JL, Campanero MR, Iglesias T. | 06/4/2011 |
| Observational study of gene-disease association, gene-environment interaction, and pharmacogenomic / toxicogenomic. (HuGE Navigator) | Variation at the NFATC2 locus increases the risk of thiazolidinedione-induced edema in the Diabetes REduction Assessment with ramipril and rosiglitazone Medication (DREAM) study.
Bailey SD, Xie C, Do R, Montpetit A, Diaz R, Mohan V, Keavney B, Yusuf S, Gerstein HC, Engert JC, Anand S, DREAM investigators., Free PMC Article | 09/15/2010 |
| Observational study of gene-disease association and gene-gene interaction. (HuGE Navigator) | Polymorphisms in innate immunity genes and risk of childhood leukemia.
Han S, Lan Q, Park AK, Lee KM, Park SK, Ahn HS, Shin HY, Kang HJ, Koo HH, Seo JJ, Choi JE, Ahn YO, Chanock SJ, Kim H, Rothman N, Kang D., Free PMC Article | 06/30/2010 |
| CCR1 antagonist, BX471, did not significantly alter ICAM-3 expression in relapsing-remitting multiple sclerosis patients. | No significant effect of orally administered chemokine receptor 1 antagonist on intercellular adhesion molecule-3 expression in relapsing--remitting multiple sclerosis patients.
Reuss R, Schreiber V, Klein A, Infante-Duarte C, Filippi M, Pabst W, Pohl C, Oschmann P. | 06/14/2010 |
| ICAM-3 enhances the migratory and invasive potential of human non-small cell lung cancer cells by inducing MMP-2 and MMP-9 via Akt and CREB | ICAM-3 enhances the migratory and invasive potential of human non-small cell lung cancer cells by inducing MMP-2 and MMP-9 via Akt and CREB.
Park JK, Park SH, So K, Bae IH, Yoo YD, Um HD. | 03/1/2010 |
| no significant risk association was found for SARS infection for the ICAM-3 Asp143Gly SNP. | Functional role of ICAM-3 polymorphism in genetic susceptibility to SARS infection.
Khoo US, Chan KY, Ching JC, Chan VS, Ip YC, Yam L, Chu CM, Lai ST, So KM, Wong TY, Chung PH, Tam P, Yip SP, Sham P, Leung GM, Lin CL, Peiris JS. | 01/25/2010 |
| Lower expression of ICAM-3 and higher expression of ICAM-1 suggest that AMs may be involved in the pathogenesis of scleroderma. | Altered adhesion molecules expression on peripheral blood mononuclear cells from patients with systemic sclerosis and clinical correlations.
Sawaya HH, de Souza RB, Carrasco S, Goldenstein-Schainberg C. | 01/21/2010 |