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    RABL3 RAB, member of RAS oncogene family like 3 [ Homo sapiens (human) ]

    Gene ID: 285282, updated on 3-Nov-2024

    GeneRIFs: Gene References Into Functions

    GeneRIFPubMed TitleDate
    Germline sequence analysis of RABL3 in a large series of pancreatic ductal adenocarcinoma patients reveals no evidence of deleterious variants.

    Germline sequence analysis of RABL3 in a large series of pancreatic ductal adenocarcinoma patients reveals no evidence of deleterious variants.
    Roberts NJ, Grant RC, Gallinger S, Klein AP, Familial Pancreatic Cancer Genome Sequencing Project., Free PMC Article

    03/12/2022
    Knockdown of RABL3 suppresses the proliferation and invasion of oral squamous cell carcinoma through inactivating the FAK/AKT pathway.

    Knockdown of RABL3 suppresses the proliferation and invasion of oral squamous cell carcinoma through inactivating the FAK/AKT pathway.
    Xu Z, Li H, Lin C, Zeng B, Chen Y, Luo Y.

    02/12/2022
    circCOL1A1 Promotes the Progression of Gastric Cancer Cells through Sponging miR-145 to Enhance RABL3 Expression.

    circCOL1A1 Promotes the Progression of Gastric Cancer Cells through Sponging miR-145 to Enhance RABL3 Expression.
    Ma Y, Ren Y, Wen H, Cui C., Free PMC Article

    02/12/2022
    Low-frequency of RABL3 pathogenetic variants in hereditary and familial pancreatic cancer.

    Low-frequency of RABL3 pathogenetic variants in hereditary and familial pancreatic cancer.
    Puzzono M, Crippa S, Zuppardo RA, Zapparoli E, Falconi M, Cavestro GM.

    01/22/2022
    Long non-coding RNA LINC00858 promotes cells proliferation and invasion through the miR-153-3p/Rabl3 axis in hepatocellular carcinoma.

    Long non-coding RNA LINC00858 promotes cells proliferation and invasion through the miR-153-3p/Rabl3 axis in hepatocellular carcinoma.
    Qi WY, Mao XB, He YB, Xiao CH.

    05/8/2021
    Identification of GIMAP7 and Rabl3 as Putative Biomarkers for Oral Squamous Cell Carcinoma Through Comparative Proteomic Approach.

    Identification of GIMAP7 and Rabl3 as Putative Biomarkers for Oral Squamous Cell Carcinoma Through Comparative Proteomic Approach.
    Usman M, Ilyas A, Hashim Z, Zarina S.

    04/13/2021
    MiR-296-3p may affect the proliferation and migration of non-small cell lung cancer cells via regulating RABL3.

    MiR-296-3p may affect the proliferation and migration of non-small cell lung cancer cells via regulating RABL3.
    Ge T, Wu HC, Zhou YY, Shen SM, Zhu LG, You GX.

    10/3/2020
    Mutations in RABL3 alter KRAS prenylation and are associated with hereditary pancreatic cancer.

    Mutations in RABL3 alter KRAS prenylation and are associated with hereditary pancreatic cancer.
    Nissim S, Leshchiner I, Mancias JD, Greenblatt MB, Maertens O, Cassa CA, Rosenfeld JA, Cox AG, Hedgepeth J, Wucherpfennig JI, Kim AJ, Henderson JE, Gonyo P, Brandt A, Lorimer E, Unger B, Prokop JW, Heidel JR, Wang XX, Ukaegbu CI, Jennings BC, Paulo JA, Gableske S, Fierke CA, Getz G, Sunyaev SR, Wade Harper J, Cichowski K, Kimmelman AC, Houvras Y, Syngal S, Williams C, Goessling W., Free PMC Article

    01/25/2020
    Hepatocellular carcinoma patients with positive expression for both Rabl3 and Cullin7 had a remarkably shorter survival time compared with patients with negative expression for both proteins.

    Overexpression of Rabl3 and Cullin7 is associated with pathogenesis and poor prognosis in hepatocellular carcinoma.
    An J, Liu Z, Liang Q, Pan Y, Li H, Wang R, Jin Y.

    10/21/2017
    Our studies suggest that the Rabl3 is a valuable marker of hepatocellular carcinoma progression and that the overexpression of Rabl3 plays an important role in the development and pathogenesis of hepatocellular carcinoma.

    The overexpression of Rabl3 is associated with pathogenesis and clinicopathologic variables in hepatocellular carcinoma.
    Pan Y, Liu Z, Feng Z, Hui D, Huang X, Tong D, Jin Y.

    06/24/2017
    We examine the correlation between the expression level of Rabl3 and survival of non-small cell lung cancer (NSCLC) patients in three independent cohorts containing 484 patients. We found high expression of Rabl3 might inhibit cell death in non-small cell lung cancer via repression of MAPK8/9/10-mediated autophagy

    High Expression of Rab-like 3 (Rabl3) is Associated with Poor Survival of Patients with Non-Small Cell Lung Cancer via Repression of MAPK8/9/10-Mediated Autophagy.
    Zhang W, Sun J, Luo J., Free PMC Article

    02/18/2017
    Rabl3 can be considered as a novel candidate in the control of tumorigenesis and as a new target for anticancer treatment.

    Evaluation of the novel gene Rabl3 in the regulation of proliferation and motility in human cancer cells.
    Li Q, Wang L, Zeng L, Zhang Y, Li K, Jin P, Su B, Wang L.

    11/13/2010
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