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NEWENTRY Record to support submission of GeneRIFs for a gene not in Gene (Vibrio cholerae El Tor N16961; Vibrio cholerae O1 biovar eltor str. N16961; Vibrio cholerae serotype O1 biotype El Tor strain N16961; Vibrio cholerae serotype O1 biotype ElTor strain N16961). [ Vibrio cholerae O1 biovar El Tor str. N16961 ]

Gene ID: 2831115, updated on 25-Jan-2023

Summary

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Gene symbol
NEWENTRY
Gene description
Record to support submission of GeneRIFs for a gene not in Gene (Vibrio cholerae El Tor N16961; Vibrio cholerae O1 biovar eltor str. N16961; Vibrio cholerae serotype O1 biotype El Tor strain N16961; Vibrio cholerae serotype O1 biotype ElTor strain N16961).
Gene type
other
Organism
Vibrio cholerae O1 biovar El Tor str. N16961
Lineage
Bacteria; Pseudomonadota; Gammaproteobacteria; Vibrionales; Vibrionaceae; Vibrio
Also known as
NEWENTRY
NEW
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Bibliography

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GeneRIFs: Gene References Into Functions

What's a GeneRIF?
  1. A PolyQ Membrane Protein of Vibrio cholerae Acts as the Receptor for Phage Infection.
    Title: A PolyQ Membrane Protein of Vibrio cholerae Acts as the Receptor for Phage Infection.
  2. Incidence of Bacterial Enteropathogens among Diarrhea Patients from Tribal Areas of Odisha.
    Title: Incidence of Bacterial Enteropathogens among Diarrhea Patients from Tribal Areas of Odisha.
  3. Inhibitors of a Na(+)-pumping NADH-ubiquinone oxidoreductase play multiple roles to block enzyme function.
    Title: Inhibitors of a Na(+)-pumping NADH-ubiquinone oxidoreductase play multiple roles to block enzyme function.
  4. Study of type IVa competence pilus of Vibrio cholerae as a model system shows that the ATPase activity of PilU is critical for pilus retraction in PilT Walker A and/or Walker B mutants. PilU does not, however, contribute to pilus retraction in DeltapilT strains. Thus, these data suggest that PilU is a bona fide retraction ATPase that supports pilus retraction in a PilT-dependent manner.[PilU, homolog of PilT]
    Title: PilT and PilU are homohexameric ATPases that coordinate to retract type IVa pili.
  5. The C4-type Zn finger motif present in the C-terminus of DksA is essential for optimal cholera toxin production. [DksA]
    Title: Post-transcriptional regulation of cholera toxin production in Vibrio cholerae by the stringent response regulator DksA.
  6. FabH1 and FabH2 protein conformation and substrate specificity.The key role of Cys356 in maintaining catalytic activity of FabH2.[FabH1][FabH2]
    Title: Differences in substrate specificity of V. cholerae FabH enzymes suggest new approaches for the development of novel antibiotics and biofuels.
  7. In vivo, ShyB alone was sufficient to sustain cell growth in low-zinc environments. In vitro, ShyB retained its d,d-endopeptidase activity against purified sacculi in the presence of the metal chelator EDTA at concentrations that inhibit ShyA and ShyC.
    Title: Endopeptidase Regulation as a Novel Function of the Zur-Dependent Zinc Starvation Response.
  8. Data suggest that AlmG, a glycyltransferase, is responsible for polymyxin resistance in this species. [AlmG, glycyltransferase-AlmG]
    Title: AlmG, responsible for polymyxin resistance in pandemic Vibrio cholerae, is a glycyltransferase distantly related to lipid A late acyltransferases.
  9. pH-dependent clcA expression is controlled by the LysR-type activator AphB, which acts in concert with AphA to initiate the virulence cascade in V. cholerae after oral ingestion. [clcA]
    Title: In vivo repressed genes of Vibrio cholerae reveal inverse requirements of an H(+)/Cl(-) transporter along the gastrointestinal passage.
  10. DksA is epistatic to HapR and RpoS and comprises a complex regulatory circuit in regulating the expression of HAP. [DksA , HapR, Hap]
    Title: DksA-HapR-RpoS axis regulates haemagglutinin protease production in Vibrio cholerae.
Submit: New GeneRIF Correction See all GeneRIFs (26)

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