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Items: 4

1.

Genome-wide analysis of primary plasma cell leukemia identifies recurrent imbalances associated with transcriptional Profile alterations

(Submitter supplied) This SuperSeries is composed of the SubSeries listed below.
Organism:
Homo sapiens
Type:
Genome variation profiling by SNP array; SNP genotyping by SNP array; Expression profiling by array
Platforms:
GPL6244 GPL3718
38 Samples
Download data: CEL
Series
Accession:
GSE39383
ID:
200039383
2.

Genome-wide analysis of primary plasma cell leukemia identifies recurrent imbalances associated with transcriptional Profile alterations (Expression)

(Submitter supplied) Primary plasma cell leukaemia (pPCL) is a rare, yet aggressive form of de novo plasma cell tumor, distinguished from secondary PCL (sPCL) which represents a leukemic transformation of pre-existing multiple myeloma (MM). Here, we performed a comprehensive molecular analysis of a prospective series of pPCLs by means of FISH, single nucleotide polymorphism (SNP) array and gene expression profiling (GEP). more...
Organism:
Homo sapiens
Type:
Expression profiling by array
Platform:
GPL6244
21 Samples
Download data: CEL
Series
Accession:
GSE39381
ID:
200039381
3.

[HuGene-1_0-st] Affymetrix Human Gene 1.0 ST Array [transcript (gene) version]

(Submitter supplied) Affymetrix submissions are typically submitted to GEO using the GEOarchive method described at http://www.ncbi.nlm.nih.gov/projects/geo/info/geo_affy.html June 03, 2009: annotation table updated with netaffx build 28 June 18, 2012: annotation table updated with netaffx build 32 July 01, 2016: annotation table updated with netaffx build 35 Protocol: See manufacturer's web site
Organism:
Homo sapiens
136 DataSets
1876 Series
27 Related Platforms
38351 Samples
Download data
Platform
Accession:
GPL6244
ID:
100006244
4.

Primary Plasma Cell Leukemia PCL-014 Expression

Organism:
Homo sapiens
Source name:
Primary Plasma Cell Leukemia PCL-014
Platform:
GPL6244
Series:
GSE39381 GSE39383
Download data: CEL
Sample
Accession:
GSM967103
ID:
300967103
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Supplemental Content

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