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Items: 4

1.

Cooperative super-enhancer inactivation caused by heterozygous loss of CREBBP and KMT2D skews B-cell fate decisions and accelerates development of T-cell depleted lymphomas.

(Submitter supplied) This SuperSeries is composed of the SubSeries listed below.
Organism:
Homo sapiens; Mus musculus
Type:
Expression profiling by high throughput sequencing; Genome binding/occupancy profiling by high throughput sequencing
Platforms:
GPL19057 GPL24676 GPL24247
143 Samples
Download data: BIGWIG, BROADPEAK, BW, MTX, NARROWPEAK, TSV
Series
Accession:
GSE224513
ID:
200224513
2.

Cooperative super-enhancer inactivation caused by heterozygous loss of CREBBP and KMT2D skews B-cell fate decisions and accelerates development of T-cell depleted lymphomas [RNA-Seq]

(Submitter supplied) Mutations in chromatin modifiers are a hallmark of many tumors, especially lymphomas arising from germinal center (GC) B cells. Given that these lymphoma mutations all induce aberrant gene repression, it is surprising that they often co-occur in individual patients. The most common pairing are mutations affecting CREBBP and KMT2D. Both impair enhancer activity in overlapping pathways to facilitate lymphomagenesis, hence their co-occurrence is especially puzzling. more...
Organism:
Homo sapiens; Mus musculus
Type:
Expression profiling by high throughput sequencing
Platforms:
GPL24676 GPL24247
53 Samples
Download data: TSV
Series
Accession:
GSE224465
ID:
200224465
3.

Illumina NovaSeq 6000 (Homo sapiens)

Platform
Accession:
GPL24676
ID:
100024676
4.

OCI-Ly7_CK_rep1

Organism:
Homo sapiens
Source name:
GCB-DLBCL
Platform:
GPL24676
Series:
GSE224465 GSE224513
Download data
Sample
Accession:
GSM7024815
ID:
307024815
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