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Items: 3

1.

Pathologically stabilized GLI3 promotes androgen-independent growth of SPOP mutant prostate cancer cells through reactivation of an AR signaling axis

(Submitter supplied) Although androgen deprivation therapy triggers a rapid response in men with metastatic prostate cancer, most patients eventually progress to lethal castration resistant prostate cancer (CRPC) characterized by androgen receptor (AR)-dependent tumor growth despite castrate levels of circulating androgens. Current treatment options for CRPC remain limited and non-curative, rendering improved outcomes dependent on deeper mechanistic insight and new therapeutic targets. more...
Organism:
Homo sapiens
Type:
Expression profiling by high throughput sequencing
Platforms:
GPL21290 GPL11154
18 Samples
Download data: TXT
Series
Accession:
GSE134682
ID:
200134682

PubMed Full text in PMC Similar studies Analyze with GEO2RSRA Run Selector

2.

Illumina HiSeq 2000 (Homo sapiens)

Organism:
Homo sapiens
10498 Series
182835 Samples
Download data
Platform
Accession:
GPL11154
ID:
100011154

3.

SPOP F102C.1 -DHT repeat 2

Organism:
Homo sapiens
Source name:
LNCaP prostate cancer cell line
Platform:
GPL11154
Series:
GSE134682
Download data: TXT
Sample
Accession:
GSM3963315
ID:
303963315

SRA Run Selector

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db=gds|term=GSM3963315[Accession]|query=1|qty=2|blobid=MCID_66e1c8c39fd03c7db383903b|ismultiple=true|min_list=5|max_list=20|def_tree=20|def_list=|def_view=|url=/Taxonomy/backend/subset.cgi?|trace_url=/stat?
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