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Items: 4

1.

Genome-wide CRISPR-Cas9 interrogation of splicing networks reveals a mechanism for recognition of autism-misregulated neuronal microexons [SPAR-seq]

(Submitter supplied) Alternative splicing has critical roles in diverse cellular, developmental and pathological processes. However, the full repertoires of factors that control individual splicing events are not known. We describe a CRISPR-based screening strategy for the systematic identification of genes that control 3-27 nt microexons with functions in nervous system development and that are commonly disrupted in autism. more...
Organism:
Mus musculus
Type:
Other
Platform:
GPL16417
48 Samples
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Series
Accession:
GSE120164
ID:
200120164
2.

Genome-wide CRISPR-Cas9 interrogation of splicing networks reveals a mechanism for recognition of autism-misregulated neuronal microexons

(Submitter supplied) This SuperSeries is composed of the SubSeries listed below.
Organism:
synthetic construct; Mus musculus
Type:
Expression profiling by high throughput sequencing; Other
4 related Platforms
122 Samples
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Series
Accession:
GSE112601
ID:
200112601
3.

Illumina MiSeq (Mus musculus)

Platform
Accession:
GPL16417
ID:
100016417
4.

SPAR-Seq of multi-exon regions of selected genes in N2A, Srrm4+siSrrm3 knockdown (T024), replicate 1

Organism:
Mus musculus
Source name:
Neuroblastoma cells
Platform:
GPL16417
Series:
GSE112601 GSE120164
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Sample
Accession:
GSM3395157
ID:
303395157
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