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Items: 3

1.

Acute TNF-induced repression of cell identity genes is mediated by NFkB-directed redistribution of cofactors from super-enhancers

(Submitter supplied) The proinflammatory cytokine tumor necrosis factor (TNF) plays a central role in low-grade adipose tissue inflammation and development of insulin resistance during obesity. In this context, nuclear factor kappa-light-chain-enhancer of activated B cells (NFkB), is directly involved and required for the acute activation of the inflammatory gene program. Here we show that the major transactivating subunit of NF?B, v-rel avian reticuloendotheliosis viral oncogene homolog A (RELA), is also required for acute TNF-induced suppression of adipocyte genes. more...
Organism:
Homo sapiens
Type:
Expression profiling by high throughput sequencing; Genome binding/occupancy profiling by high throughput sequencing
Platform:
GPL18460
36 Samples
Download data: BW, TXT
2.

Illumina HiSeq 1500 (Homo sapiens)

Platform
Accession:
GPL18460
ID:
100018460
3.

BRD4 ChIP-seq Vehicle

Organism:
Homo sapiens
Source name:
Adipocyte
Platform:
GPL18460
Series:
GSE64233
Download data: BW
Sample
Accession:
GSM1709938
ID:
301709938
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db=gds|term=GSM1709938[Accession]|query=1|qty=2|blobid=MCID_664fa06bd01e38019ca2bfb3|ismultiple=true|min_list=5|max_list=20|def_tree=20|def_list=|def_view=|url=/Taxonomy/backend/subset.cgi?|trace_url=/stat?
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