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Items: 4

1.

Neuropathy of haematopoietic stem cell niche is essential for myeloproliferative neoplasms

(Submitter supplied) This SuperSeries is composed of the SubSeries listed below.
Organism:
Mus musculus
Type:
Expression profiling by high throughput sequencing; Expression profiling by array
Platforms:
GPL6246 GPL11002
6 Samples
Download data: CEL
Series
Accession:
GSE55802
ID:
200055802
2.

Sympathetic neuropathy of the bone marrow haematopoietic stem cell niche is essential for myeloproliferative neoplasms

(Submitter supplied) Myeloproliferative neoplasms (MPNs) are diseases caused by mutations in the haematopoietic stem cell (HSC) compartment. Most MPN patients have a common acquired mutation of Janus kinase 2 (JAK2) gene in HSCs that renders this kinase constitutively active, leading to uncontrolled cell expansion. The bone marrow (BM) microenvironment might contribute to the clinical outcomes of this common event. We previously showed that BM nestin+ mesenchymal stem cells (MSCs) innervated by sympathetic nerve fibres regulate normal HSCs. more...
Organism:
Mus musculus
Type:
Expression profiling by array
Platform:
GPL6246
4 Samples
Download data: CEL
Series
Accession:
GSE55801
ID:
200055801
3.

[MoGene-1_0-st] Affymetrix Mouse Gene 1.0 ST Array [transcript (gene) version]

(Submitter supplied) Affymetrix submissions are typically submitted to GEO using the GEOarchive method described at http://www.ncbi.nlm.nih.gov/projects/geo/info/geo_affy.html June 03, 2009: annotation table updated with netaffx build 28 June 07, 2012: annotation table updated with netaffx build 32 July 01, 2016: annotation table updated with netaffx build 35 Protocol: See manufacturer's web site
Organism:
Mus musculus
143 DataSets
2508 Series
28 Related Platforms
30473 Samples
Download data
Platform
Accession:
GPL6246
ID:
100006246
4.

Control mouse

Organism:
Mus musculus
Source name:
Bone marrow stromal nestin+ cells, control
Platform:
GPL6246
Series:
GSE55801 GSE55802
Download data: CEL
Sample
Accession:
GSM1346232
ID:
301346232
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Supplemental Content

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