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Items: 6

1.

Nucleotide stress induction of HEXIM1 suppresses melanoma by modulating cancer cell-specific gene transcription [reChIP-seq]

(Submitter supplied) Cancer metabolism has been actively studied to gain insights into tumorigenic survival mechanisms and susceptibilities. In melanoma, we identify HEXIM1, a transcription elongation regulator, as a novel melanoma suppressor that participates in nucleotide stress regulation. HEXIM1 expression is low in melanoma. Its overexpression suppresses melanoma while its inactivation accelerates tumor onset in vivo. more...
Organism:
Homo sapiens
Type:
Genome binding/occupancy profiling by high throughput sequencing
Platform:
GPL16791
4 Samples
Download data: BEDGRAPH
Series
Accession:
GSE68047
ID:
200068047
2.

Illumina HiSeq 2500 (Homo sapiens)

Platform
Accession:
GPL16791
ID:
100016791
3.

HEXIM1>CDK9 reChIP on A375 cells

Organism:
Homo sapiens
Source name:
A375 melanoma cell line
Platform:
GPL16791
Series:
GSE68047 GSE68053
Download data
Sample
Accession:
GSM1661558
ID:
301661558
4.

Input reChIP on A375 cells (DMSO HEXIM1-CDK9 reChIP Input)

Organism:
Homo sapiens
Source name:
A375 melanoma cell line
Platform:
GPL16791
Series:
GSE68047 GSE68053
Download data
Sample
Accession:
GSM1661557
ID:
301661557
5.

CDK9>HEXIM1 reChIP on A375 cells

Organism:
Homo sapiens
Source name:
A375 melanoma cell line
Platform:
GPL16791
Series:
GSE68047 GSE68053
Download data
Sample
Accession:
GSM1661556
ID:
301661556
6.

Input reChIP on A375 cells (DMSO CDK9-HEXIM1 reChIP Input)

Organism:
Homo sapiens
Source name:
A375 melanoma cell line
Platform:
GPL16791
Series:
GSE68047 GSE68053
Download data
Sample
Accession:
GSM1661555
ID:
301661555
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Supplemental Content

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